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10.1016/j.semnephrol.2006.07.001 http://scihub22266oqcxt.onion/10.1016/j.semnephrol.2006.07.001 17071327!2878276!17071327     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Semin+Nephrol 2006 ; 26 (5): 334-44 Nephropedia Template TP {{tp|p=17071327|t=2006. Na+/H+ exchangers in renal regulation of acid-base balance |pdf=|usr=}}{{17071327}} gab.com Text Na+/H+ exchangers in renal regulation of acid-base balance JO: Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=17071327 #FOAvip The kidney plays key roles in extracellular fluid pH homeostasis by reclaiming bicarbonate (HCO(3)(-)) filtered at the glomerulus and generating the consumed HCO(3)(-) by secreting protons (H(+)) into the urine (renal acidification). Sodium-proton exchangers (NHEs) are ubiquitous transmembrane proteins mediating the countertransport of Na(+) and H(+) across lipid bilayers. In mammals, NHEs participate in the regulation of cell pH, volume, and intracellular sodium concentration, as well as in transepithelial ion transport. Five of the 10 isoforms (NHE1-4 and NHE8) are expressed at the plasma membrane of renal epithelial cells. The best-studied isoform for acid-base homeostasis is NHE3, which mediates both HCO(3)(-) absorption and H(+) excretion in the renal tubule. This article reviews some important aspects of NHEs in the kidney, with special emphasis on the role of renal NHE3 in the maintenance of acid-base balance. Twit Text FOAVip Na+/H+ exchangers in renal regulation of acid-base balance ????Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=17071327 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=17071327 #FOAvip English Wikipedia <ref>{{Cite pmid|17071327}}</ref> Na+/H+ exchangers in renal regulation of acid-base balance #MMPMID17071327 Bobulescu IA; Moe OW Semin Nephrol 2006[Sep]; 26 (5): 334-44 PMID17071327show ga The kidney plays key roles in extracellular fluid pH homeostasis by reclaiming bicarbonate (HCO(3)(-)) filtered at the glomerulus and generating the consumed HCO(3)(-) by secreting protons (H(+)) into the urine (renal acidification). Sodium-proton exchangers (NHEs) are ubiquitous transmembrane proteins mediating the countertransport of Na(+) and H(+) across lipid bilayers. In mammals, NHEs participate in the regulation of cell pH, volume, and intracellular sodium concentration, as well as in transepithelial ion transport. Five of the 10 isoforms (NHE1-4 and NHE8) are expressed at the plasma membrane of renal epithelial cells. The best-studied isoform for acid-base homeostasis is NHE3, which mediates both HCO(3)(-) absorption and H(+) excretion in the renal tubule. This article reviews some important aspects of NHEs in the kidney, with special emphasis on the role of renal NHE3 in the maintenance of acid-base balance. |Acid-Base Equilibrium/*physiology[MESH] |Acidosis/physiopathology[MESH] |Adaptation, Physiological[MESH] |Animals[MESH] |Humans[MESH] |Nephrons/*physiology[MESH]Na+/H+ exchangers in renal regulation of acid-base balance (epmc).pdf DeepDyve Pubget Overpricing