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10.1016/j.virol.2006.03.029 http://scihub22266oqcxt.onion/10.1016/j.virol.2006.03.029 16643977!ä!16643977 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Virology 2006 ; 349 (2): 254-63 Nephropedia Template TP {{tp|p=16643977|t=2006. Survival of parvovirus B19-infected cells by cellular autophagy |pdf=|usr=}}{{16643977}} gab.com Text Survival of parvovirus B19-infected cells by cellular autophagy JO: Virology http://www.kidney.de/pget.php?&tw=1&pmid=16643977 #FOAvip Human parvovirus B19 (B19) is a well-known pathogenic agent which causes apoptosis in erythrocyte lineage cells. Here, we provide the first evidence that mitochondrial autophagy is specifically found in the B19-infected cells. The protein expression ratio for LC3-II/LC3-I increased significantly in infected cells, indicating possible involvement of cellular autophagy in the infection process. Immunofluorescence confocal microscopy analyses revealed that B19 infection induced an intracellular autophagosome as judged by endogenous LC3 staining. Moreover, inhibition of autophagy by 3-MA significantly facilitated B19-infection-mediated cell death. These results suggest a novel mechanism by which B19-infected cells survive by cellular autophagy. Twit Text FOAVip Survival of parvovirus B19-infected cells by cellular autophagy ????Virology http://www.kidney.de/pget.php?&tw=1&pmid=16643977 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=16643977 #FOAvip English Wikipedia <ref>{{Cite pmid|16643977}}</ref> Survival of parvovirus B19-infected cells by cellular autophagy #MMPMID16643977 Nakashima A; Tanaka N; Tamai K; Kyuuma M; Ishikawa Y; Sato H; Yoshimori T; Saito S; Sugamura K Virology 2006[Jun]; 349 (2): 254-63 PMID16643977show ga Human parvovirus B19 (B19) is a well-known pathogenic agent which causes apoptosis in erythrocyte lineage cells. Here, we provide the first evidence that mitochondrial autophagy is specifically found in the B19-infected cells. The protein expression ratio for LC3-II/LC3-I increased significantly in infected cells, indicating possible involvement of cellular autophagy in the infection process. Immunofluorescence confocal microscopy analyses revealed that B19 infection induced an intracellular autophagosome as judged by endogenous LC3 staining. Moreover, inhibition of autophagy by 3-MA significantly facilitated B19-infection-mediated cell death. These results suggest a novel mechanism by which B19-infected cells survive by cellular autophagy. |Adenine/analogs & derivatives/pharmacology[MESH] |Autophagy/*physiology[MESH] |Blotting, Western[MESH] |Cell Line, Tumor[MESH] |Cell Survival[MESH] |Cytoplasm/chemistry/ultrastructure[MESH] |Enzyme Inhibitors/pharmacology[MESH] |Erythroid Cells/ultrastructure/*virology[MESH] |Flow Cytometry[MESH] |Humans[MESH] |Microscopy, Fluorescence[MESH] |Microtubule-Associated Proteins/analysis[MESH] |Parvovirus B19, Human/*physiology[MESH] |Phagosomes/chemistry/ultrastructure[MESH]Survival of parvovirus B19-infected cells by cellular autophagy (epmc).pdf DeepDyve Pubget Overpricing