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10.1007/s11010-005-2733-9 http://scihub22266oqcxt.onion/10.1007/s11010-005-2733-9 16180088!ä!16180088 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=16180088&cmd=llinks): Failed to open stream: HTTP request failed! 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Intestinal and cardiac inflammatory response shows enhanced endotoxin receptor (CD14) expression in magnesium deficiency |pdf=|usr=}}{{16180088}} gab.com Text Intestinal and cardiac inflammatory response shows enhanced endotoxin receptor (CD14) expression in magnesium deficiency JO: Mol Cell Biochem http://www.kidney.de/pget.php?&tw=1&pmid=16180088 #FOAvip Substance P is elevated in plasma and in other tissues during Mg-deficiency, and was found localised to neuronal C-fibres of cardiac and intestinal tissues, where it could promote neurogenic inflammation. Plasma prostaglandin E2 (PGE2), indicative of systemic inflammation, rose significantly (>or=4 fold, p<0.01) after 1 week and remained elevated through week 2 and 3 in rat on the Mg-deficient (MgD) diet. Concomitantly, total blood glutathione decreased by 50%. Immunohistochemical staining for endotoxin (lipopolysaccaride, LPS) receptor, CD14 was prominent in macrophage-type cells in intestinal tissue; more importantly, cardiac tissue revealed both CD11b (monocyte/macrophage surface protein) and CD14 positive cells after 3 weeks in rats on MgD diet. Western blot analysis indicated a significant increase in the endotoxin receptor protein level in the 3 week MgD hearts. Since CD14 is known to be up-regulated in cells exposed to LPS, these observations suggest that prolonged Mg-deficiency results in increased intestinal permeability to bacterial products that induce the endotoxin receptor in cells localized to myocardial and intestinal tissues. These CD14 positive cells may amplify the cardiomyopathic inflammatory process by stimulating TNF-alpha and other pro-inflammatory cytokines. Twit Text FOAVip Intestinal and cardiac inflammatory response shows enhanced endotoxin receptor (CD14) expression in magnesium deficiency ????Mol Cell Biochem http://www.kidney.de/pget.php?&tw=1&pmid=16180088 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=16180088 #FOAvip English Wikipedia <ref>{{Cite pmid|16180088}}</ref> Intestinal and cardiac inflammatory response shows enhanced endotoxin receptor (CD14) expression in magnesium deficiency #MMPMID16180088 Chmielinska JJ; Tejero-Taldo MI; Mak IT; Weglicki WB Mol Cell Biochem 2005[Oct]; 278 (1-2): 53-7 PMID16180088show ga Substance P is elevated in plasma and in other tissues during Mg-deficiency, and was found localised to neuronal C-fibres of cardiac and intestinal tissues, where it could promote neurogenic inflammation. Plasma prostaglandin E2 (PGE2), indicative of systemic inflammation, rose significantly (>or=4 fold, p<0.01) after 1 week and remained elevated through week 2 and 3 in rat on the Mg-deficient (MgD) diet. Concomitantly, total blood glutathione decreased by 50%. Immunohistochemical staining for endotoxin (lipopolysaccaride, LPS) receptor, CD14 was prominent in macrophage-type cells in intestinal tissue; more importantly, cardiac tissue revealed both CD11b (monocyte/macrophage surface protein) and CD14 positive cells after 3 weeks in rats on MgD diet. Western blot analysis indicated a significant increase in the endotoxin receptor protein level in the 3 week MgD hearts. Since CD14 is known to be up-regulated in cells exposed to LPS, these observations suggest that prolonged Mg-deficiency results in increased intestinal permeability to bacterial products that induce the endotoxin receptor in cells localized to myocardial and intestinal tissues. These CD14 positive cells may amplify the cardiomyopathic inflammatory process by stimulating TNF-alpha and other pro-inflammatory cytokines. |Animals[MESH] |Fluorescent Antibody Technique[MESH] |Inflammation/*metabolism[MESH] |Intestinal Mucosa/metabolism[MESH] |Intestines/cytology[MESH] |Magnesium Deficiency/*complications[MESH] |Male[MESH] |Myocardium/cytology/metabolism[MESH] |Rats[MESH] |Rats, Sprague-Dawley[MESH] |Receptors, Immunologic/blood/*metabolism[MESH] |Species Specificity[MESH]Intestinal and cardiac inflammatory response shows enhanced endotoxin (epmc).pdf DeepDyve Pubget Overpricing