Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText.
Kick-your-searchterm to multiple Engines kick-your-query now !>
A dictionary by aggregated review articles of nephrology, medicine and the life sciences
Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

suck abstract from ncbi


10.1681/ASN.V210s190

http://scihub22266oqcxt.onion/10.1681/ASN.V210s190
suck pdf from google scholar
1600136!ä!1600136

suck abstract from ncbi


Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534
pmid1600136      J+Am+Soc+Nephrol 1992 ; 2 (10 Suppl): S190-7
Nephropedia Template TP

gab.com Text

Twit Text FOAVip

Twit Text #

English Wikipedia


  • The activated mesangial cell: a glomerular "myofibroblast"? #MMPMID1600136
  • Johnson RJ; Floege J; Yoshimura A; Iida H; Couser WG; Alpers CE
  • J Am Soc Nephrol 1992[Apr]; 2 (10 Suppl): S190-7 PMID1600136show ga
  • The glomerular mesangial cell may have several important beneficial functions in the normal glomerulus. These include the production of growth factors to allow normal cell turnover, the provision of structural support for the capillaries via the production of mesangial matrix, and the modulation of glomerular hemodynamics via their contractile properties. However, in various types of glomerular injury, the mesangial cell may acquire characteristics of a "myofibroblast", which may in fact be injurious to the glomerulus. These "activated" mesangial cells can be shown to be proliferating by one or more mechanisms that are mediated by platelets and that also involve the local production of platelet-derived growth factor. Like myofibroblasts in other tissues, the mesangial cell acquires smooth muscle cell-like properties, characterized by the de novo expression of alpha-smooth muscle actin, and by the development of fibroblast-like properties, characterized by the production of interstitial collagens in addition to normal mesangial matrix constituents. Identifying therapeutic strategies that prevent this phenotypic modulation of the mesangial cell may provide new ways to treat golmerular diseases.
  • |Animals[MESH]
  • |Blood Platelets/physiology[MESH]
  • |Cell Division[MESH]
  • |Complement System Proteins/physiology[MESH]
  • |Fibroblasts/cytology[MESH]
  • |Glomerular Mesangium/*cytology/physiology[MESH]
  • |Glomerulonephritis/etiology/pathology[MESH]
  • |Growth Substances/physiology[MESH]
  • |Humans[MESH]


  • DeepDyve
  • Pubget Overpricing
  • suck abstract from ncbi

    Linkout box