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10.1016/j.virol.2004.08.019 http://scihub22266oqcxt.onion/10.1016/j.virol.2004.08.019 15518807!?!15518807 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=15518807&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Virology 2004 ; 329 (2): 270-9 Nephropedia Template TP {{tp|p=15518807|t=2004. No apoptotic deaths and different levels of inductions of inflammatory cytokines in alveolar macrophages infected with influenza viruses |pdf=|usr=}}{{15518807}} gab.com Text No apoptotic deaths and different levels of inductions of inflammatory cytokines in alveolar macrophages infected with influenza viruses JO: Virology http://www.kidney.de/pget.php?&tw=1&pmid=15518807 #FOAvip Influenza viruses are reported to infect mainly the respiratory tract epithelium of hosts. Our studies in a pig model show that influenza A viruses infect alveolar macrophages that constitutively reside in the respiratory tract, without causing apoptosis. Tumor necrosis factor alpha was the inflammatory cytokine most highly induced in these macrophages. In vivo, alveolar macrophages infected with human H3N2 influenza virus showed greater expression of tumor necrosis factor alpha than did alveolar macrophages infected with human H1N1 influenza virus. Induction of specific inflammatory cytokine such as TNF-alpha is a polygenic trait that involves the HA and NA genes. Markedly elevated expression of tumor necrosis factor alpha may be responsible for the high mortality rate caused by H3N2 influenza virus infection in elderly patients. Twit Text FOAVip No apoptotic deaths and different levels of inductions of inflammatory cytokines in alveolar macrophages infected with influenza viruses ????Virology http://www.kidney.de/pget.php?&tw=1&pmid=15518807 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=15518807 #FOAvip English Wikipedia <ref>{{Cite pmid|15518807}}</ref> No apoptotic deaths and different levels of inductions of inflammatory cytokines in alveolar macrophages infected with influenza viruses #MMPMID15518807 Seo SH; Webby R; Webster RG Virology 2004[Nov]; 329 (2): 270-9 PMID15518807show ga Influenza viruses are reported to infect mainly the respiratory tract epithelium of hosts. Our studies in a pig model show that influenza A viruses infect alveolar macrophages that constitutively reside in the respiratory tract, without causing apoptosis. Tumor necrosis factor alpha was the inflammatory cytokine most highly induced in these macrophages. In vivo, alveolar macrophages infected with human H3N2 influenza virus showed greater expression of tumor necrosis factor alpha than did alveolar macrophages infected with human H1N1 influenza virus. Induction of specific inflammatory cytokine such as TNF-alpha is a polygenic trait that involves the HA and NA genes. Markedly elevated expression of tumor necrosis factor alpha may be responsible for the high mortality rate caused by H3N2 influenza virus infection in elderly patients. |*Apoptosis[MESH] |Animals[MESH] |Cells, Cultured[MESH] |Cytokines/analysis/*biosynthesis[MESH] |Disease Models, Animal[MESH] |Influenza A virus/genetics/*pathogenicity[MESH] |Influenza B virus/*pathogenicity[MESH] |Macrophages, Alveolar/*immunology/pathology/virology[MESH] |Orthomyxoviridae Infections/blood/*immunology[MESH] |Respiratory System/*immunology[MESH] |Swine[MESH] |Time Factors[MESH]No apoptotic deaths and different levels of inductions of inflammatory(epmc).pdf DeepDyve Pubget Overpricing