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10.1097/01.pec.0000117927.65522.7a http://scihub22266oqcxt.onion/10.1097/01.pec.0000117927.65522.7a 15094576!ä!15094576 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Pediatr+Emerg+Care 2004 ; 20 (3): 175-177 Nephropedia Template TP {{tp|p=15094576|t=2004. Catecholaminergic polymorphic ventricular tachycardia: successful emergency treatment with intravenous propranolol |pdf=|usr=}}{{15094576}} gab.com Text Catecholaminergic polymorphic ventricular tachycardia: successful emergency treatment with intravenous propranolol JO: Pediatr Emerg Care http://www.kidney.de/pget.php?&tw=1&pmid=15094576 #FOAvip Catecholaminergic polymorphic ventricular tachycardia (VT) is a rare arrhythmogenic disorder, which may cause sudden death and whose relationships with mutations in cardiac ryanodine receptor gene have been recently established. The present article reports a catecholaminergic polymorphic VT case of a 9-year-old girl, without any previous history of syncope, who has been found unconscious while playing and referred comatose to pediatric intensive care unit. The electrocardiogram pattern showed runs of bidirectional and polymorphic VT degenerating into ventricular fibrillation, without QT interval abnormalities. Various attempts of cardioversion, lidocaine, and magnesium sulfate intravenous infusions were only partially effective. Owing to catecholaminergic polymorphic VT highly suggesting electrocardiogram pattern, intravenous propranolol was administered, achieving immediate VT interruption. Long-term nadolol therapy effectively prevented further arrhythmias, with no relapses up to 10 months later; a good neurologic recovery was also obtained. Genetic evaluation revealed in this patient-but not in relatives-a mutation in ryanodine receptor gene on chromosome 1. Twit Text FOAVip Catecholaminergic polymorphic ventricular tachycardia: successful emergency treatment with intravenous propranolol ????Pediatr Emerg Care http://www.kidney.de/pget.php?&tw=1&pmid=15094576 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=15094576 #FOAvip English Wikipedia <ref>{{Cite pmid|15094576}}</ref> Catecholaminergic polymorphic ventricular tachycardia: successful emergency treatment with intravenous propranolol #MMPMID15094576 De Rosa G; Delogu AB; Piastra M; Chiaretti A; Bloise R; Priori SG Pediatr Emerg Care 2004[Mar]; 20 (3): 175-177 PMID15094576show ga Catecholaminergic polymorphic ventricular tachycardia (VT) is a rare arrhythmogenic disorder, which may cause sudden death and whose relationships with mutations in cardiac ryanodine receptor gene have been recently established. The present article reports a catecholaminergic polymorphic VT case of a 9-year-old girl, without any previous history of syncope, who has been found unconscious while playing and referred comatose to pediatric intensive care unit. The electrocardiogram pattern showed runs of bidirectional and polymorphic VT degenerating into ventricular fibrillation, without QT interval abnormalities. Various attempts of cardioversion, lidocaine, and magnesium sulfate intravenous infusions were only partially effective. Owing to catecholaminergic polymorphic VT highly suggesting electrocardiogram pattern, intravenous propranolol was administered, achieving immediate VT interruption. Long-term nadolol therapy effectively prevented further arrhythmias, with no relapses up to 10 months later; a good neurologic recovery was also obtained. Genetic evaluation revealed in this patient-but not in relatives-a mutation in ryanodine receptor gene on chromosome 1. |Adrenergic beta-Antagonists/administration & dosage/*therapeutic use[MESH] |Brain Edema/etiology[MESH] |Catecholamines/metabolism[MESH] |Child[MESH] |Decerebrate State[MESH] |Electrocardiography[MESH] |Emergencies[MESH] |Female[MESH] |Humans[MESH] |Infusions, Intravenous[MESH] |Intensive Care Units, Pediatric[MESH] |Nadolol/therapeutic use[MESH] |Propranolol/administration & dosage/*therapeutic use[MESH] |Ryanodine Receptor Calcium Release Channel/genetics[MESH] |Syncope/etiology[MESH] |Tachycardia, Ventricular/diagnosis/diagnostic imaging/*drug therapy/genetics[MESH]Catecholaminergic polymorphic ventricular tachycardia: successful emer(epmc).pdf DeepDyve Pubget Overpricing