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http://scihub22266oqcxt.onion/ 14989640!ä!14989640 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biotech+Histochem 2003 ; 78 (5): 231-6 Nephropedia Template TP {{tp|p=14989640|t=2003. Alterations in osteoclast morphology following osteoprotegerin administration in the magnesium-deficient mouse |pdf=|usr=}}{{14989640}} gab.com Text Alterations in osteoclast morphology following osteoprotegerin administration in the magnesium-deficient mouse JO: Biotech Histochem http://www.kidney.de/pget.php?&tw=1&pmid=14989640 #FOAvip In the present study, we used osteoprotegerin (OPG), which blocks osteoclastogenesis, to correct and thus explain the hypercalcemia that is seen during dietary Mg deficiency in the mouse. Control and Mg-deficient mice received injections for 12 days of either OPG or vehicle only. Serum Ca was similar in Mg-deficient mice treated with OPG and in control mice receiving OPG (9.2 +/- 0.3 mg/dl vs. 9.2 +/- 0.5). Both groups had significantly higher serum Ca than controls or Mg-deficient animals receiving vehicle alone. Surprisingly, Mg-depleted mice that received OPG in doses that inhibit osteoclastic bone resorption remained hypercalcemic. Because mature osteoclasts still present in the marrow might be hyperactive, we examined osteoclast morphology at the light microscopic and ultrastructural level. Light microscopic examination of trabecular bone showed few osteoclasts in OPG-treated mice. Ultrastructural examination revealed that osteoclasts in OPG-treated mice have decreased contact with the endosteal bone surface and absence of a ruffled border. Because the morphology of the existing pool of mature osteoclasts did not enhance resorption, another mechanism, such as increased intestinal absorption of Ca in Mg-deficient mice, likely contributes to the hypercalcemia observed during Mg deficiency. Twit Text FOAVip Alterations in osteoclast morphology following osteoprotegerin administration in the magnesium-deficient mouse ????Biotech Histochem http://www.kidney.de/pget.php?&tw=1&pmid=14989640 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=14989640 #FOAvip English Wikipedia <ref>{{Cite pmid|14989640}}</ref> Alterations in osteoclast morphology following osteoprotegerin administration in the magnesium-deficient mouse #MMPMID14989640 Gruber HE; Rude RK Biotech Histochem 2003[Oct]; 78 (5): 231-6 PMID14989640show ga In the present study, we used osteoprotegerin (OPG), which blocks osteoclastogenesis, to correct and thus explain the hypercalcemia that is seen during dietary Mg deficiency in the mouse. Control and Mg-deficient mice received injections for 12 days of either OPG or vehicle only. Serum Ca was similar in Mg-deficient mice treated with OPG and in control mice receiving OPG (9.2 +/- 0.3 mg/dl vs. 9.2 +/- 0.5). Both groups had significantly higher serum Ca than controls or Mg-deficient animals receiving vehicle alone. Surprisingly, Mg-depleted mice that received OPG in doses that inhibit osteoclastic bone resorption remained hypercalcemic. Because mature osteoclasts still present in the marrow might be hyperactive, we examined osteoclast morphology at the light microscopic and ultrastructural level. Light microscopic examination of trabecular bone showed few osteoclasts in OPG-treated mice. Ultrastructural examination revealed that osteoclasts in OPG-treated mice have decreased contact with the endosteal bone surface and absence of a ruffled border. Because the morphology of the existing pool of mature osteoclasts did not enhance resorption, another mechanism, such as increased intestinal absorption of Ca in Mg-deficient mice, likely contributes to the hypercalcemia observed during Mg deficiency. |Acid Phosphatase/analysis[MESH] |Animals[MESH] |Calcium/blood[MESH] |Female[MESH] |Glycoproteins/*pharmacology[MESH] |Hypercalcemia/*etiology[MESH] |Isoenzymes/analysis[MESH] |Magnesium Deficiency/*complications/pathology[MESH] |Mice[MESH] |Osteoclasts/drug effects/*pathology/ultrastructure[MESH] |Osteoprotegerin[MESH] |Receptors, Cytoplasmic and Nuclear[MESH] |Receptors, Tumor Necrosis Factor[MESH]Alterations in osteoclast morphology following osteoprotegerin adminis(epmc).pdf DeepDyve Pubget Overpricing