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10.1016/S0140-6736(04)15595-5 http://scihub22266oqcxt.onion/10.1016/S0140-6736(04)15595-5 14987888!7112424!14987888     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Lancet 2004 ; 363 (9409): 617-9 Nephropedia Template TP {{tp|p=14987888|t=2004. Re-emergence of fatal human influenza A subtype H5N1 disease |pdf=|usr=}}{{14987888}} gab.com Text Re-emergence of fatal human influenza A subtype H5N1 disease JO: Lancet http://www.kidney.de/pget.php?&tw=1&pmid=14987888 #FOAvip Human disease associated with influenza A subtype H5N1 re-emerged in January, 2003, for the first time since an outbreak in Hong Kong in 1997. Patients with H5N1 disease had unusually high serum concentrations of chemokines (eg, interferon induced protein-10 [IP-10] and monokine induced by interferon gamma [MIG]). Taken together with a previous report that H5N1 influenza viruses induce large amounts of proinflammatory cytokines from macrophage cultures in vitro, our findings suggest that cytokine dysfunction contributes to the pathogenesis of H5N1 disease. Development of vaccines against influenza A (H5N1) virus should be made a priority. Twit Text FOAVip Re-emergence of fatal human influenza A subtype H5N1 disease ????Lancet http://www.kidney.de/pget.php?&tw=1&pmid=14987888 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=14987888 #FOAvip English Wikipedia <ref>{{Cite pmid|14987888}}</ref> Re-emergence of fatal human influenza A subtype H5N1 disease #MMPMID14987888 Peiris JS; Yu WC; Leung CW; Cheung CY; Ng WF; Nicholls JM; Ng TK; Chan KH; Lai ST; Lim WL; Yuen KY; Guan Y Lancet 2004[Feb]; 363 (9409): 617-9 PMID14987888show ga Human disease associated with influenza A subtype H5N1 re-emerged in January, 2003, for the first time since an outbreak in Hong Kong in 1997. Patients with H5N1 disease had unusually high serum concentrations of chemokines (eg, interferon induced protein-10 [IP-10] and monokine induced by interferon gamma [MIG]). Taken together with a previous report that H5N1 influenza viruses induce large amounts of proinflammatory cytokines from macrophage cultures in vitro, our findings suggest that cytokine dysfunction contributes to the pathogenesis of H5N1 disease. Development of vaccines against influenza A (H5N1) virus should be made a priority. |Animals[MESH] |China/epidemiology[MESH] |Disease Outbreaks/statistics & numerical data[MESH] |Hong Kong/epidemiology[MESH] |Humans[MESH] |Influenza A Virus, H5N1 Subtype[MESH] |Influenza A virus/isolation & purification[MESH] |Influenza in Birds/*epidemiology/transmission/virology[MESH] |Influenza, Human/*epidemiology/*transmission/virology[MESH] |Poultry[MESH] |Poultry Diseases/epidemiology/transmission[MESH]Re-emergence of fatal human influenza A subtype H5N1 disease (epmc).pdf DeepDyve Pubget Overpricing