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10.1203/01.PDR.0000117848.37520.A2 http://scihub22266oqcxt.onion/10.1203/01.PDR.0000117848.37520.A2 14764909!ä!14764909 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=14764909&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Pediatr+Res 2004 ; 55 (5): 734-7 Nephropedia Template TP {{tp|p=14764909|t=2004. Dual-function ion channel/protein kinases: novel components of vertebrate magnesium regulatory mechanisms |pdf=|usr=}}{{14764909}} gab.com Text Dual-function ion channel/protein kinases: novel components of vertebrate magnesium regulatory mechanisms JO: Pediatr Res http://www.kidney.de/pget.php?&tw=1&pmid=14764909 #FOAvip Although magnesium is the dominant divalent intracellular cation and is required for the function of diverse types of enzymes that participate in virtually every cellular process, the molecular mechanisms that regulate its homeostasis are poorly understood. Electrophysiologic and biochemical investigations of a novel dual-function ion channel/kinase protein have recently converged with the identification of the gene locus for an autosomal recessive form of inherited hypomagnesemia to provide new insight into vertebrate magnesium regulatory mechanisms. Twit Text FOAVip Dual-function ion channel/protein kinases: novel components of vertebrate magnesium regulatory mechanisms ????Pediatr Res http://www.kidney.de/pget.php?&tw=1&pmid=14764909 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=14764909 #FOAvip English Wikipedia <ref>{{Cite pmid|14764909}}</ref> Dual-function ion channel/protein kinases: novel components of vertebrate magnesium regulatory mechanisms #MMPMID14764909 Schmitz C; Perraud AL; Fleig A; Scharenberg AM Pediatr Res 2004[May]; 55 (5): 734-7 PMID14764909show ga Although magnesium is the dominant divalent intracellular cation and is required for the function of diverse types of enzymes that participate in virtually every cellular process, the molecular mechanisms that regulate its homeostasis are poorly understood. Electrophysiologic and biochemical investigations of a novel dual-function ion channel/kinase protein have recently converged with the identification of the gene locus for an autosomal recessive form of inherited hypomagnesemia to provide new insight into vertebrate magnesium regulatory mechanisms. |Animals[MESH] |Biological Transport[MESH] |Cations[MESH] |Cations, Divalent[MESH] |Electrophysiology[MESH] |Humans[MESH] |Ion Channels/*chemistry/*physiology[MESH] |Magnesium/chemistry[MESH] |Membrane Proteins/*physiology[MESH] |Models, Biological[MESH] |Protein Kinases/*chemistry/*physiology[MESH] |Protein Serine-Threonine Kinases[MESH]Dual-function ion channel/protein kinases: novel components of vertebr(epmc).pdf DeepDyve Pubget Overpricing