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10.1016/S0022-2143(03)00062-3

http://scihub22266oqcxt.onion/10.1016/S0022-2143(03)00062-3
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12960953!?!12960953

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suck abstract from ncbi

pmid12960953      J+Lab+Clin+Med 2003 ; 142 (2): 71-82
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  • Aldosteronism revisited: perspectives on less well-recognized actions of aldosterone #MMPMID12960953
  • Weber KT
  • J Lab Clin Med 2003[Aug]; 142 (2): 71-82 PMID12960953show ga
  • Aldosterone is a mineralocorticoid with protean actions in both epithelial and nonepithelial cells. These include endocrine properties of circulating aldosterone that promote Na(+) resorption at the expense of well-recognized K(+) excretion and less well-recognized Mg(2+) excretion in classic target tissues: kidneys, colon, and sweat and salivary glands. The regulation of adrenal aldosterone secretion by [Mg(2+)](o) is also less well appreciated. More recently recognized endocrine actions of aldosterone include induction of Mg(2+) efflux in exchange for Na(+) in such nonepithelial cells as peripheral-blood mononuclear cells and influence on epithelial cells of the choroid plexus, where aldosterone alters the composition of cerebrospinal fluid that contributes to blood-pressure regulation. An association between primary aldosteronism and idiopathic intracranial hypertension has recently been reported. Extraadrenal steroidogenesis with de novo aldosterone production by the cardiovasculature, where its auto-/paracrine properties may contribute to tissue repair at sites of injury, has been observed. These less well-recognized actions of aldosterone have led to a revival of interest in how this steroid molecule contributes to the pathophysiology of various clinical disorders.
  • |Aldosterone/*physiology[MESH]
  • |Animals[MESH]
  • |Humans[MESH]


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