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10.1354/vp.40-3-304 http://scihub22266oqcxt.onion/10.1354/vp.40-3-304 12724572!ä!12724572 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=12724572&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Vet+Pathol 2003 ; 40 (3): 304-10 Nephropedia Template TP {{tp|p=12724572|t=2003. Pathology of human influenza A (H5N1) virus infection in cynomolgus macaques (Macaca fascicularis) |pdf=|usr=}}{{12724572}} gab.com Text Pathology of human influenza A (H5N1) virus infection in cynomolgus macaques (Macaca fascicularis) JO: Vet Pathol http://www.kidney.de/pget.php?&tw=1&pmid=12724572 #FOAvip Infection with influenza A (H5N1) virus, which has not been associated with respiratory disease in humans previously, caused clinical signs of acute respiratory distress syndrome and multiple-organ dysfunction syndrome with high mortality in humans in Hong Kong in 1997. To study the pathogenesis of this disease, we infected four cynomolgus monkeys (Macaca fascicularis) with 2.5 x 104 median tissue culture infectious dose (TCID50) of influenza virus A/Hong Kong/156/97 (H5N1) and euthanatized them 4 or 7 days after infection. The main lesion was a necrotizing broncho-interstitial pneumonia (4/4) similar to those found in primary influenza virus pneumonia in humans, with desquamation of respiratory epithelium (4/4), intra-alveolar hemorrhage (4/4), hyaline membrane formation (2/4), and infiltration with neutrophils and macrophages (4/4). Lesions in other organs consisted of a suppurative tonsillitis (2/4) and necrosis in lymphoid organs (1/4), kidney (1/4), and liver (1/4). By immunohistochemistry, influenza virus antigen was limited to pulmonary tissue (4/4) and tonsils (2/4). Based on these results, we suggest that the cynomolgus monkey is a suitable animal model for studying the pathogenesis of human H5N1 virus infection and that multiple-organ dysfunction syndrome in this disease may be caused by diffuse alveolar damage from virus replication in the lungs alone. Twit Text FOAVip Pathology of human influenza A (H5N1) virus infection in cynomolgus macaques (Macaca fascicularis) ????Vet Pathol http://www.kidney.de/pget.php?&tw=1&pmid=12724572 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=12724572 #FOAvip English Wikipedia <ref>{{Cite pmid|12724572}}</ref> Pathology of human influenza A (H5N1) virus infection in cynomolgus macaques (Macaca fascicularis) #MMPMID12724572 Kuiken T; Rimmelzwaan GF; Van Amerongen G; Osterhaus AD Vet Pathol 2003[May]; 40 (3): 304-10 PMID12724572show ga Infection with influenza A (H5N1) virus, which has not been associated with respiratory disease in humans previously, caused clinical signs of acute respiratory distress syndrome and multiple-organ dysfunction syndrome with high mortality in humans in Hong Kong in 1997. To study the pathogenesis of this disease, we infected four cynomolgus monkeys (Macaca fascicularis) with 2.5 x 104 median tissue culture infectious dose (TCID50) of influenza virus A/Hong Kong/156/97 (H5N1) and euthanatized them 4 or 7 days after infection. The main lesion was a necrotizing broncho-interstitial pneumonia (4/4) similar to those found in primary influenza virus pneumonia in humans, with desquamation of respiratory epithelium (4/4), intra-alveolar hemorrhage (4/4), hyaline membrane formation (2/4), and infiltration with neutrophils and macrophages (4/4). Lesions in other organs consisted of a suppurative tonsillitis (2/4) and necrosis in lymphoid organs (1/4), kidney (1/4), and liver (1/4). By immunohistochemistry, influenza virus antigen was limited to pulmonary tissue (4/4) and tonsils (2/4). Based on these results, we suggest that the cynomolgus monkey is a suitable animal model for studying the pathogenesis of human H5N1 virus infection and that multiple-organ dysfunction syndrome in this disease may be caused by diffuse alveolar damage from virus replication in the lungs alone. |*Influenza A Virus, H5N1 Subtype[MESH] |*Macaca fascicularis[MESH] |Animals[MESH] |Antigens, Viral/metabolism[MESH] |Female[MESH] |Humans[MESH] |Immunohistochemistry/veterinary[MESH] |Influenza A virus/*growth & development[MESH] |Influenza, Human/*pathology/*veterinary/virology[MESH] |Lung/pathology/virology[MESH] |Male[MESH] |Monkey Diseases/*pathology/*virology[MESH]Pathology of human influenza A (H5N1) virus infection in cynomolgus ma(epmc).pdf DeepDyve Pubget Overpricing