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10.1099/0022-1317-83-2-273 http://scihub22266oqcxt.onion/10.1099/0022-1317-83-2-273 11807219!ä!11807219 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Gen+Virol 2002 ; 83 (Pt 2): 273-281 Nephropedia Template TP {{tp|p=11807219|t=2002. A transgenic mouse model for non-immune hydrops fetalis induced by the NS1 gene of human parvovirus B19 |pdf=|usr=}}{{11807219}} gab.com Text A transgenic mouse model for non-immune hydrops fetalis induced by the NS1 gene of human parvovirus B19 JO: J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=11807219 #FOAvip Human parvovirus B19 (B19) infection during pregnancy is associated with the adverse foetal outcome known as non-immune hydrops fetalis (NIHF). Although B19 is known to infect erythroid-lineage cells in vivo as well as in vitro, the mechanism leading to the occurrence of NIHF is not clear. To investigate the possible involvement of the B19 non-structural protein NS1 in NIHF, three independent lines of transgenic mice were generated that expressed NS1 under the control of the Cre-loxP system and the GATA1 promoter. Two of the three lines expressed NS1 in erythroid-lineage cells. Most of the transgenic mice died at the embryonic stage, some of which developed hydropic changes caused by severe anaemia at embryonic day 15.5 (E15.5). Histological examination of embryos at E15.5 showed significantly fewer erythropoietic islands in the liver parenchyma, whereas their hearts showed no abnormal signs, such as cardiomegaly and apoptotic cells. The NS1-transgenic mouse lines established here provide an animal model for human NIHF and suggest that NS1 plays a crucial role in the adverse outcome associated with intrauterine B19 infection in humans. Twit Text FOAVip A transgenic mouse model for non-immune hydrops fetalis induced by the NS1 gene of human parvovirus B19 ????J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=11807219 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=11807219 #FOAvip English Wikipedia <ref>{{Cite pmid|11807219}}</ref> A transgenic mouse model for non-immune hydrops fetalis induced by the NS1 gene of human parvovirus B19 #MMPMID11807219 Chisaka H; Morita E; Murata K; Ishii N; Yaegashi N; Okamura K; Sugamura K J Gen Virol 2002[Feb]; 83 (Pt 2): 273-281 PMID11807219show ga Human parvovirus B19 (B19) infection during pregnancy is associated with the adverse foetal outcome known as non-immune hydrops fetalis (NIHF). Although B19 is known to infect erythroid-lineage cells in vivo as well as in vitro, the mechanism leading to the occurrence of NIHF is not clear. To investigate the possible involvement of the B19 non-structural protein NS1 in NIHF, three independent lines of transgenic mice were generated that expressed NS1 under the control of the Cre-loxP system and the GATA1 promoter. Two of the three lines expressed NS1 in erythroid-lineage cells. Most of the transgenic mice died at the embryonic stage, some of which developed hydropic changes caused by severe anaemia at embryonic day 15.5 (E15.5). Histological examination of embryos at E15.5 showed significantly fewer erythropoietic islands in the liver parenchyma, whereas their hearts showed no abnormal signs, such as cardiomegaly and apoptotic cells. The NS1-transgenic mouse lines established here provide an animal model for human NIHF and suggest that NS1 plays a crucial role in the adverse outcome associated with intrauterine B19 infection in humans. |*Disease Models, Animal[MESH] |*Mice, Transgenic[MESH] |Animals[MESH] |DNA-Binding Proteins/genetics/metabolism[MESH] |Embryo, Mammalian/pathology[MESH] |Erythroid Precursor Cells[MESH] |Erythroid-Specific DNA-Binding Factors[MESH] |Female[MESH] |Fetal Death[MESH] |GATA1 Transcription Factor[MESH] |Hydrops Fetalis/embryology/*etiology/pathology[MESH] |Integrases/genetics/metabolism[MESH] |Mice[MESH] |Parvoviridae Infections/*complications/pathology/physiopathology[MESH] |Parvovirus B19, Human/*genetics/metabolism[MESH] |Pregnancy[MESH] |Pregnancy Complications, Infectious/pathology/physiopathology[MESH] |Transcription Factors/genetics/metabolism[MESH] |Tumor Cells, Cultured[MESH] |Viral Nonstructural Proteins/*genetics/metabolism[MESH]A transgenic mouse model for non-immune hydrops fetalis induced by the(epmc).pdf DeepDyve Pubget Overpricing