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10.1007/s005350170061

http://scihub22266oqcxt.onion/10.1007/s005350170061
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11519837!ä!11519837

suck abstract from ncbi


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pmid11519837      J+Gastroenterol 2001 ; 36 (8): 564-8
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  • Alcoholic liver cirrhosis complicated with torsade de pointes during plasma exchange and hemodiafiltration #MMPMID11519837
  • Nakasone H; Sugama R; Sakugawa H; Matayoshi R; Miyagi T; Maeshiro T; Yamashiro T; Higa F; Hokama A; Kinjo F; Saito A; Toda T
  • J Gastroenterol 2001[Aug]; 36 (8): 564-8 PMID11519837show ga
  • A 36-year-old man with severe alcoholic hepatitis was treated with plasma exchange combined with hemodiafiltration to remove endotoxins and inflammatory cytokines. During the treatment, he had critical arrhythmia (torsade de pointes [TdP]). His laboratory data showed hypomagnesemia, which was suspected to be responsible for the development of TdP. Patients with alcoholic liver disease tend to have hypomagnesemia and Q-T interval prolongation. Furthermore, hemodiafiltration may cause hypomagnesemia. Careful observation for electrolytic imbalance is necessary when clinicians treat patients with alcoholic liver failure with a liver support system.
  • |Adult[MESH]
  • |Electrocardiography/methods[MESH]
  • |Hemodiafiltration/*adverse effects[MESH]
  • |Humans[MESH]
  • |Liver Cirrhosis, Alcoholic/blood/complications/*therapy[MESH]
  • |Magnesium/*blood[MESH]
  • |Male[MESH]
  • |Plasma Exchange/*adverse effects[MESH]


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