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10.1016/s0264-410x(00)00275-9 http://scihub22266oqcxt.onion/10.1016/s0264-410x(00)00275-9 11163460!ä!11163460       Vaccine 2000 ; 19 Suppl 1 (ä): S32-7 Nephropedia Template TP {{tp|p=11163460|t=2000. Inflammatory responses in influenza A virus infection |pdf=|usr=}}{{11163460}} gab.com Text Inflammatory responses in influenza A virus infection JO: Vaccine http://www.kidney.de/pget.php?&tw=1&pmid=11163460 #FOAvip Influenza A virus causes respiratory tract infections, which are occasionally complicated by secondary bacterial infections. Influenza A virus replicates in epithelial cells and leukocytes resulting in the production of chemokines and cytokines, which favor the extravasation of blood mononuclear cells and the development of antiviral and Th1-type immune response. Influenza A virus-infected respiratory epithelial cells produce limited amounts of chemokines (RANTES, MCP-1, IL-8) and IFN-alpha/beta, whereas monocytes/macrophages readily produce chemokines such as RANTES, MIP-1alpha, MCP-1, MCP-3, IP-10 and cytokines TNF-alpha, IL-1beta, IL-6, IL-18 and IFN-alpha/beta. The role of influenza A virus-induced inflammatory response in relation to otitis media is being discussed. Twit Text FOAVip Inflammatory responses in influenza A virus infection ????Vaccine http://www.kidney.de/pget.php?&tw=1&pmid=11163460 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=11163460 #FOAvip English Wikipedia <ref>{{Cite pmid|11163460}}</ref> Inflammatory responses in influenza A virus infection #MMPMID11163460 Julkunen I; Melen K; Nyqvist M; Pirhonen J; Sareneva T; Matikainen S Vaccine 2000[Dec]; 19 Suppl 1 (ä): S32-7 PMID11163460show ga Influenza A virus causes respiratory tract infections, which are occasionally complicated by secondary bacterial infections. Influenza A virus replicates in epithelial cells and leukocytes resulting in the production of chemokines and cytokines, which favor the extravasation of blood mononuclear cells and the development of antiviral and Th1-type immune response. Influenza A virus-infected respiratory epithelial cells produce limited amounts of chemokines (RANTES, MCP-1, IL-8) and IFN-alpha/beta, whereas monocytes/macrophages readily produce chemokines such as RANTES, MIP-1alpha, MCP-1, MCP-3, IP-10 and cytokines TNF-alpha, IL-1beta, IL-6, IL-18 and IFN-alpha/beta. The role of influenza A virus-induced inflammatory response in relation to otitis media is being discussed. |Apoptosis[MESH] |Bacterial Infections[MESH] |Cytokines/*physiology[MESH] |Epithelium/microbiology/virology[MESH] |Gene Expression Regulation, Viral[MESH] |Humans[MESH] |Inflammation[MESH] |Influenza A virus/*physiology[MESH] |Influenza, Human/*immunology[MESH] |Interferon-alpha/physiology[MESH] |Macrophages/physiology[MESH] |Models, Biological[MESH] |Otitis Media/immunology[MESH] |Superinfection[MESH] |Th1 Cells/immunology[MESH] |Transcription Factors/physiology[MESH] |Transcription, Genetic[MESH]Inflammatory responses in influenza A virus infection (epmc).pdf DeepDyve Pubget Overpricing