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10.1111/j.1750-3639.2000.tb00241.x http://scihub22266oqcxt.onion/10.1111/j.1750-3639.2000.tb00241.x 10668894!8098463!10668894     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Brain+Pathol 2000 ; 10 (1): 39-48 Nephropedia Template TP {{tp|p=10668894|t=2000. Distribution of Borna disease virus in the brain of rats infected with an obesity-inducing virus strain |pdf=|usr=}}{{10668894}} gab.com Text Distribution of Borna disease virus in the brain of rats infected with an obesity-inducing virus strain JO: Brain Pathol http://www.kidney.de/pget.php?&tw=1&pmid=10668894 #FOAvip Experimental infection of Lewis rats with Borna disease virus (BDV), a nonsegmented, single-stranded RNA virus, usually causes an immune-mediated biphasic neurobehavioral disorder. Such animals develop a persistent infection of the CNS with viral antigen expression in all brain regions and a disseminated nonpurulent meningoencephalitis. Interestingly, intracerebral infection of Lewis rats with a BDV-variant (BDV-ob) causes a rapid increase of body weight with the development of an obesity syndrome without obvious neurological signs. The obese phenotype is correlated with a characteristic distribution of inflammatory lesions and BDV-antigen in the rat brain. Infiltration with mononuclear immune cells and viral antigen expression are restricted to the septum, hippocampus, amygdala and ventromedian tuberal hypothalamus. Therefore, infection with the obesity-inducing BDV-ob results most likely in neuroendocrine dysregulations leading to the development of an obesity syndrome. This might be due to the restriction of viral antigen expression and inflammatory lesions to brain areas which are involved in the regulation of body weight and food intake. The BDV-induced obesity syndrome represents a model for the study of immune-mediated neuroendocrine disorders caused by viral infections of the CNS. Twit Text FOAVip Distribution of Borna disease virus in the brain of rats infected with an obesity-inducing virus strain ????Brain Pathol http://www.kidney.de/pget.php?&tw=1&pmid=10668894 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=10668894 #FOAvip English Wikipedia <ref>{{Cite pmid|10668894}}</ref> Distribution of Borna disease virus in the brain of rats infected with an obesity-inducing virus strain #MMPMID10668894 Herden C; Herzog S; Richt JA; Nesseler A; Christ M; Failing K; Frese K Brain Pathol 2000[Jan]; 10 (1): 39-48 PMID10668894show ga Experimental infection of Lewis rats with Borna disease virus (BDV), a nonsegmented, single-stranded RNA virus, usually causes an immune-mediated biphasic neurobehavioral disorder. Such animals develop a persistent infection of the CNS with viral antigen expression in all brain regions and a disseminated nonpurulent meningoencephalitis. Interestingly, intracerebral infection of Lewis rats with a BDV-variant (BDV-ob) causes a rapid increase of body weight with the development of an obesity syndrome without obvious neurological signs. The obese phenotype is correlated with a characteristic distribution of inflammatory lesions and BDV-antigen in the rat brain. Infiltration with mononuclear immune cells and viral antigen expression are restricted to the septum, hippocampus, amygdala and ventromedian tuberal hypothalamus. Therefore, infection with the obesity-inducing BDV-ob results most likely in neuroendocrine dysregulations leading to the development of an obesity syndrome. This might be due to the restriction of viral antigen expression and inflammatory lesions to brain areas which are involved in the regulation of body weight and food intake. The BDV-induced obesity syndrome represents a model for the study of immune-mediated neuroendocrine disorders caused by viral infections of the CNS. |Animals[MESH] |Borna Disease/*pathology[MESH] |Borna disease virus/*metabolism[MESH] |Brain/*pathology/*virology[MESH] |Immunohistochemistry[MESH] |Obesity/*pathology/*virology[MESH] |Rats[MESH]Distribution of Borna disease virus in the brain of rats infected with(epmc).pdf DeepDyve Pubget Overpricing