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10.1097/00045415-199811000-00007 http://scihub22266oqcxt.onion/10.1097/00045415-199811000-00007 10348955!ä!10348955 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cardiol+Rev 1998 ; 6 (6): 319-324 Nephropedia Template TP {{tp|p=10348955|t=1998. Acquired QT Prolongation: Mechanisms and Implications |pdf=|usr=}}{{10348955}} gab.com Text Acquired QT Prolongation: Mechanisms and Implications JO: Cardiol Rev http://www.kidney.de/pget.php?&tw=1&pmid=10348955 #FOAvip QT interval prolongation can be caused by several drugs. Torsade de pointes (TdP) is a form of ventricular proarrhythmia associated with QT interval prolongation, and in some cases, the development of U waves. The mechanism of TdP is believed to be related to an abnormality of repolarization in the ventricular myocardium and the development of early afterdepolarizations. QT prolongation leading to TdP can be caused by antiarrhythmic drugs (primarily class IA and III drugs), as well as a variety of other drugs. These other classes of drugs include phenothiazines, tricyclic antidepressants, antihistamines, antibiotics, antifungal agents, and antianginals. The recognition of a patient profile at increased risk for the development of TdP can help the patient avoid this drug-related complication. Treatment of TdP depends on the correct identification of the problem and discontinuation of the agent causing TdP, as well as correction of metabolic abnormalities, increase in the resting heart rate, and infusion of magnesium. Prompt diagnosis and treatment of this condition are central to avoid poor outcome. Twit Text FOAVip Acquired QT Prolongation: Mechanisms and Implications ????Cardiol Rev http://www.kidney.de/pget.php?&tw=1&pmid=10348955 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=10348955 #FOAvip English Wikipedia <ref>{{Cite pmid|10348955}}</ref> Acquired QT Prolongation: Mechanisms and Implications #MMPMID10348955 Fu EY; Clemo HF; Ellenbogen KA Cardiol Rev 1998[Nov]; 6 (6): 319-324 PMID10348955show ga QT interval prolongation can be caused by several drugs. Torsade de pointes (TdP) is a form of ventricular proarrhythmia associated with QT interval prolongation, and in some cases, the development of U waves. The mechanism of TdP is believed to be related to an abnormality of repolarization in the ventricular myocardium and the development of early afterdepolarizations. QT prolongation leading to TdP can be caused by antiarrhythmic drugs (primarily class IA and III drugs), as well as a variety of other drugs. These other classes of drugs include phenothiazines, tricyclic antidepressants, antihistamines, antibiotics, antifungal agents, and antianginals. The recognition of a patient profile at increased risk for the development of TdP can help the patient avoid this drug-related complication. Treatment of TdP depends on the correct identification of the problem and discontinuation of the agent causing TdP, as well as correction of metabolic abnormalities, increase in the resting heart rate, and infusion of magnesium. Prompt diagnosis and treatment of this condition are central to avoid poor outcome. äAcquired QT Prolongation: Mechanisms and Implications (epmc).pdf DeepDyve Pubget Overpricing