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10.1097/FJC.0000000000000206 http://scihub22266oqcxt.onion/10.1097/FJC.0000000000000206 C4355281!4355281!25551320     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25551320&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25551320.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Cardiovasc+Pharmacol 2015 ; 65 (3): 218-25 Nephropedia Template TP {{tp|p=25551320|t=2015. mAKAP ? A Master Scaffold for Cardiac Remodeling |pdf=|usr=}}{{25551320}} gab.com Text mAKAP A Master Scaffold for Cardiac Remodeling JO: J Cardiovasc Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=25551320 #FOAvip Cardiac remodeling is regulated by an extensive intracellular signal transduction network. Each of the many signaling pathways in this network contributes uniquely to the control of cellular adaptation. In the last few years, it has become apparent that multimolecular signaling complexes or ?signalosomes? are important for fidelity in intracellular signaling and for mediating crosstalk between the different signaling pathways. These complexes integrate upstream signals and control downstream effectors. In the cardiac myocyte, the protein mAKAP? serves as a scaffold for a large signalosome that is responsive to cAMP, calcium, hypoxia, and mitogen-activated protein kinase signaling. The main function of mAKAP? signalosomes is to modulate stress-related gene expression regulated by the transcription factors NFATc, MEF2 and HIF-1? and type II histone deacetylases that control pathological cardiac hypertrophy. Twit Text FOAVip mAKAP A Master Scaffold for Cardiac Remodeling ????J Cardiovasc Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=25551320 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25551320 #FOAvip English Wikipedia <ref>{{Cite pmid|25551320}}</ref> mAKAP ? A Master Scaffold for Cardiac Remodeling #MMPMID25551320 Passariello CL; Li J; Dodge-Kafka K; Kapiloff MS J Cardiovasc Pharmacol 2015[Mar]; 65 (3): 218-25 PMID25551320show ga Cardiac remodeling is regulated by an extensive intracellular signal transduction network. Each of the many signaling pathways in this network contributes uniquely to the control of cellular adaptation. In the last few years, it has become apparent that multimolecular signaling complexes or ?signalosomes? are important for fidelity in intracellular signaling and for mediating crosstalk between the different signaling pathways. These complexes integrate upstream signals and control downstream effectors. In the cardiac myocyte, the protein mAKAP? serves as a scaffold for a large signalosome that is responsive to cAMP, calcium, hypoxia, and mitogen-activated protein kinase signaling. The main function of mAKAP? signalosomes is to modulate stress-related gene expression regulated by the transcription factors NFATc, MEF2 and HIF-1? and type II histone deacetylases that control pathological cardiac hypertrophy. ämAKAP ? A Master Scaffold for Cardiac Remodeling (epmc).pdf DeepDyve Pubget Overpricing