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10.3389/fphys.2018.00762 http://scihub22266oqcxt.onion/10.3389/fphys.2018.00762 C6018397!6018397 !29971019     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29971019 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Front+Physiol 2018 ; 9 (ä): 762 Nephropedia Template TP {{tp|p=29971019 |t=2018. Vitamin C in Cancer: A Metabolomics Perspective |pdf=|usr=}}{{29971019 }} gab.com Text Vitamin C in Cancer: A Metabolomics Perspective JO: Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=29971019 #FOAvip There is an ongoing interest in cellular antioxidants and oxidants as well as cellular mechanisms underlying their effects. Several reports suggest that vitamin C (L-ascorbic acid) functions as a pro-oxidant with selective toxicity against specific types of tumor cells. In addition, reduced glutathione plays an emerging role in reducing oxidative stress due to xenobiotic toxins such as metals and oxidants associated with diseases such as cancer, cardiovascular disease, and stroke. High-dose intravenous vitamin C and intravenous glutathione have been used as complementary, alternative, and adjuvant medicines. Here, we review the molecular mechanisms underlying the regulation of oxidation/reduction systems, focusing on the altered metabolomics profile in cancer cells following treatment with pharmacological vitamin C. This review focuses on the role of vitamin C in energy metabolism in terms of adenosine triphosphate, cysteine, and reduced glutathione levels, affecting cancer cell death. Twit Text FOAVip Vitamin C in Cancer: A Metabolomics Perspective ????Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=29971019 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29971019 #FOAvip English Wikipedia <ref>{{Cite pmid|29971019 }}</ref> Vitamin C in Cancer: A Metabolomics Perspective #MMPMID29971019 Park S ; Ahn S ; Shin Y ; Yang Y ; Yeom CH Front Physiol 2018[]; 9 (ä): 762 PMID29971019 show ga There is an ongoing interest in cellular antioxidants and oxidants as well as cellular mechanisms underlying their effects. Several reports suggest that vitamin C (L-ascorbic acid) functions as a pro-oxidant with selective toxicity against specific types of tumor cells. In addition, reduced glutathione plays an emerging role in reducing oxidative stress due to xenobiotic toxins such as metals and oxidants associated with diseases such as cancer, cardiovascular disease, and stroke. High-dose intravenous vitamin C and intravenous glutathione have been used as complementary, alternative, and adjuvant medicines. Here, we review the molecular mechanisms underlying the regulation of oxidation/reduction systems, focusing on the altered metabolomics profile in cancer cells following treatment with pharmacological vitamin C. This review focuses on the role of vitamin C in energy metabolism in terms of adenosine triphosphate, cysteine, and reduced glutathione levels, affecting cancer cell death. äVitamin C in Cancer: A Metabolomics Perspective (epmc).pdf DeepDyve Pubget Overpricing