Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24819870
&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests
in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215
Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24819870
.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117 Curr+Infect+Dis+Rep
2014 ; 16
(6
): 407
Nephropedia Template TP
gab.com Text
Twit Text FOAVip
Twit Text #
English Wikipedia
Update on varicella zoster virus vasculopathy
#MMPMID24819870
Nagel MA
; Gilden D
Curr Infect Dis Rep
2014[Jun]; 16
(6
): 407
PMID24819870
show ga
Primary infection of humans with varicella zoster virus (VZV) causes varicella
(chickenpox), after which the virus becomes latent in cranial nerve ganglia,
dorsal root ganglia and autonomic ganglia along the entire neuraxis. As
VZV-specific cell-mediated immunity declines in elderly and immunocompromised
individuals, VZV reactivates from one or more ganglia and typically causes herpes
zoster (shingles). Zoster may also be complicated by VZV vasculopathy due to
productive virus infection of the cerebral arteries. In recent decades, the
clinical spectrum of VZV vasculopathy has expanded to include not only transient
ischemic attacks and ischemic and hemorrhagic stroke, but also multifocal VZV
vasculopathy, with temporal artery infection mimicking giant cell arteritis,
extracranial vasculopathy, aneurysm with and without subarachnoid hemorrhage,
arterial dissection and dolichoectasia, ischemic cranial neuropathies, cerebral
venous sinus thrombosis, spinal cord infarction and peripheral thrombotic
disease.