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10.1586/14779072.2015.1074861 http://scihub22266oqcxt.onion/10.1586/14779072.2015.1074861 C4829576!4829576!26308600     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26308600&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26308600.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Expert+Rev+Cardiovasc+Ther 2015 ; 13 (9): 975-87 Nephropedia Template TP {{tp|p=26308600|t=2015. Understanding the pathogenesis of abdominal aortic aneurysms |pdf=|usr=}}{{26308600}} gab.com Text Understanding the pathogenesis of abdominal aortic aneurysms JO: Expert Rev Cardiovasc Ther http://www.kidney.de/pget.php?&tw=1&pmid=26308600 #FOAvip An aortic aneurysm is a dilatation in which the aortic diameter is ? 3.0 cm. If left untreated, the aortic wall continues to weaken and becomes unable to withstand the forces of the luminal blood pressure resulting in progressive dilatation and rupture, a catastrophic event associated with a mortality of 50 ? 80%. Smoking and positive family history are important risk factors for the development of abdominal aortic aneurysms (AAA). Several genetic risk factors have also been identified. On the histological level, visible hallmarks of AAA pathogenesis include inflammation, smooth muscle cell apoptosis, extracellular matrix degradation, and oxidative stress. We expect that large genetic, genomic, epigenetic, proteomic and metabolomic studies will be undertaken by international consortia to identify additional risk factors and biomarkers, and to enhance our understanding of the pathobiology of AAA. Collaboration between different research groups will be important in overcoming the challenges to develop pharmacological treatments for AAA. Twit Text FOAVip Understanding the pathogenesis of abdominal aortic aneurysms ????Expert Rev Cardiovasc Ther http://www.kidney.de/pget.php?&tw=1&pmid=26308600 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26308600 #FOAvip English Wikipedia <ref>{{Cite pmid|26308600}}</ref> Understanding the pathogenesis of abdominal aortic aneurysms #MMPMID26308600 Kuivaniemi H; Ryer EJ; Elmore JR; Tromp G Expert Rev Cardiovasc Ther 2015[]; 13 (9): 975-87 PMID26308600show ga An aortic aneurysm is a dilatation in which the aortic diameter is ? 3.0 cm. If left untreated, the aortic wall continues to weaken and becomes unable to withstand the forces of the luminal blood pressure resulting in progressive dilatation and rupture, a catastrophic event associated with a mortality of 50 ? 80%. Smoking and positive family history are important risk factors for the development of abdominal aortic aneurysms (AAA). Several genetic risk factors have also been identified. On the histological level, visible hallmarks of AAA pathogenesis include inflammation, smooth muscle cell apoptosis, extracellular matrix degradation, and oxidative stress. We expect that large genetic, genomic, epigenetic, proteomic and metabolomic studies will be undertaken by international consortia to identify additional risk factors and biomarkers, and to enhance our understanding of the pathobiology of AAA. Collaboration between different research groups will be important in overcoming the challenges to develop pharmacological treatments for AAA. äUnderstanding the pathogenesis of abdominal aortic aneurysms (epmc).pdf DeepDyve Pubget Overpricing