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10.4239/wjd.v6.i12.1207 http://scihub22266oqcxt.onion/10.4239/wjd.v6.i12.1207 C4598604!4598604 !26464759     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26464759 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       World+J+Diabetes 2015 ; 6 (12 ): 1207-22 Nephropedia Template TP {{tp|p=26464759 |t=2015. Treatment of prediabetes |pdf=|usr=}}{{26464759 }} gab.com Text Treatment of prediabetes JO: World J Diabetes http://www.kidney.de/pget.php?&tw=1&pmid=26464759 #FOAvip Progression of normal glucose tolerance (NGT) to overt diabetes is mediated by a transition state called impaired glucose tolerance (IGT). Beta cell dysfunction and insulin resistance are the main defects in type 2 diabetes mellitus (type 2 DM) and even normoglycemic IGT patients manifest these defects. Beta cell dysfunction and insulin resistance also contribute to the progression of IGT to type 2 DM. Improving insulin sensitivity and/or preserving functions of beta-cells can be a rational way to normalize the GT and to control transition of IGT to type 2 DM. Loosing weight, for example, improves whole body insulin sensitivity and preserves beta-cell function and its inhibitory effect on progression of IGT to type 2 DM had been proven. But interventions aiming weight loss usually not applicable in real life. Pharmacotherapy is another option to gain better insulin sensitivity and to maintain beta-cell function. In this review, two potential treatment options (lifestyle modification and pharmacologic agents) that limits the IGT-type 2 DM conversion in prediabetic subjects are discussed. Twit Text FOAVip Treatment of prediabetes ????World J Diabetes http://www.kidney.de/pget.php?&tw=1&pmid=26464759 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26464759 #FOAvip English Wikipedia <ref>{{Cite pmid|26464759 }}</ref> Treatment of prediabetes #MMPMID26464759 Kanat M ; DeFronzo RA ; Abdul-Ghani MA World J Diabetes 2015[Sep]; 6 (12 ): 1207-22 PMID26464759 show ga Progression of normal glucose tolerance (NGT) to overt diabetes is mediated by a transition state called impaired glucose tolerance (IGT). Beta cell dysfunction and insulin resistance are the main defects in type 2 diabetes mellitus (type 2 DM) and even normoglycemic IGT patients manifest these defects. Beta cell dysfunction and insulin resistance also contribute to the progression of IGT to type 2 DM. Improving insulin sensitivity and/or preserving functions of beta-cells can be a rational way to normalize the GT and to control transition of IGT to type 2 DM. Loosing weight, for example, improves whole body insulin sensitivity and preserves beta-cell function and its inhibitory effect on progression of IGT to type 2 DM had been proven. But interventions aiming weight loss usually not applicable in real life. Pharmacotherapy is another option to gain better insulin sensitivity and to maintain beta-cell function. In this review, two potential treatment options (lifestyle modification and pharmacologic agents) that limits the IGT-type 2 DM conversion in prediabetic subjects are discussed. äTreatment of prediabetes (epmc).pdf DeepDyve Pubget Overpricing