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10.1101/cshperspect.a016261 http://scihub22266oqcxt.onion/10.1101/cshperspect.a016261 C4413233!4413233 !25213094     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25213094 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25213094 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Cold+Spring+Harb+Perspect+Biol 2014 ; 6 (11 ): a016261 Nephropedia Template TP {{tp|p=25213094 |t=2014. Transcriptional control of inflammatory responses |pdf=|usr=}}{{25213094 }} gab.com Text Transcriptional control of inflammatory responses JO: Cold Spring Harb Perspect Biol http://www.kidney.de/pget.php?&tw=1&pmid=25213094 #FOAvip The inflammatory response requires the activation of a complex transcriptional program that is both cell-type- and stimulus-specific and involves the dynamic regulation of hundreds of genes. In the context of an inflamed tissue, extensive changes in gene expression occur in both parenchymal cells and infiltrating cells of the immune system. Recently, basic transcriptional mechanisms that control inflammation have been clarified at a genome scale, particularly in macrophages and conventional dendritic cells. The regulatory logic of distinct groups of inflammatory genes can be explained to some extent by identifiable sequence-encoded features of their chromatin organization, which impact on transcription factor (TF) accessibility and impose different requirements for gene activation. Moreover, it has become apparent that the interplay between TFs activated by inflammatory stimuli and master regulators exerts a crucial role in controlling cell-type-specific transcriptional outputs. Twit Text FOAVip Transcriptional control of inflammatory responses ????Cold Spring Harb Perspect Biol http://www.kidney.de/pget.php?&tw=1&pmid=25213094 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25213094 #FOAvip English Wikipedia <ref>{{Cite pmid|25213094 }}</ref> Transcriptional control of inflammatory responses #MMPMID25213094 Smale ST ; Natoli G Cold Spring Harb Perspect Biol 2014[Sep]; 6 (11 ): a016261 PMID25213094 show ga The inflammatory response requires the activation of a complex transcriptional program that is both cell-type- and stimulus-specific and involves the dynamic regulation of hundreds of genes. In the context of an inflamed tissue, extensive changes in gene expression occur in both parenchymal cells and infiltrating cells of the immune system. Recently, basic transcriptional mechanisms that control inflammation have been clarified at a genome scale, particularly in macrophages and conventional dendritic cells. The regulatory logic of distinct groups of inflammatory genes can be explained to some extent by identifiable sequence-encoded features of their chromatin organization, which impact on transcription factor (TF) accessibility and impose different requirements for gene activation. Moreover, it has become apparent that the interplay between TFs activated by inflammatory stimuli and master regulators exerts a crucial role in controlling cell-type-specific transcriptional outputs. |*Gene Expression Regulation [MESH] |Chromatin/metabolism [MESH] |CpG Islands/immunology [MESH] |Dendritic Cells/immunology/metabolism [MESH] |Inflammation/*genetics/immunology [MESH] |Macrophages/immunology/metabolism [MESH] |Nucleosomes/immunology/metabolism/physiology [MESH]Transcriptional control of inflammatory responses (epmc).pdf DeepDyve Pubget Overpricing