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10.1159/000365149 http://scihub22266oqcxt.onion/10.1159/000365149 C4476411!4476411 !25195856     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25195856 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Vasc+Res 2014 ; 51 (4 ): 247-58 Nephropedia Template TP {{tp|p=25195856 |t=2014. The role of pericyte detachment in vascular rarefaction |pdf=|usr=}}{{25195856 }} gab.com Text The role of pericyte detachment in vascular rarefaction JO: J Vasc Res http://www.kidney.de/pget.php?&tw=1&pmid=25195856 #FOAvip BACKGROUND: Pericytes surround endothelial cells at the perivascular interface. Signaling between endothelial cells and pericytes is crucial for capillary homeostasis, as pericytes stabilize vessels and regulate many microvascular functions. Recently it has been shown that pericytes are able to detach from the vascular wall and contribute to fibrosis by becoming scar-forming myofibroblasts in many organs including the kidney. At the same time, the loss of pericytes within the perivascular compartment results in vulnerable capillaries which are prone to instability, pathological angiogenesis, and, ultimately, rarefaction. AIMS: This review will give an overview of pericyte-endothelial cell interactions, summarize the signaling pathways that have been identified to be involved in pericyte detachment from the vascular wall, and present pathological endothelial responses in the context of disease of the kidney. Twit Text FOAVip The role of pericyte detachment in vascular rarefaction ????J Vasc Res http://www.kidney.de/pget.php?&tw=1&pmid=25195856 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25195856 #FOAvip English Wikipedia <ref>{{Cite pmid|25195856 }}</ref> The role of pericyte detachment in vascular rarefaction #MMPMID25195856 Schrimpf C ; Teebken OE ; Wilhelmi M ; Duffield JS J Vasc Res 2014[]; 51 (4 ): 247-58 PMID25195856 show ga BACKGROUND: Pericytes surround endothelial cells at the perivascular interface. Signaling between endothelial cells and pericytes is crucial for capillary homeostasis, as pericytes stabilize vessels and regulate many microvascular functions. Recently it has been shown that pericytes are able to detach from the vascular wall and contribute to fibrosis by becoming scar-forming myofibroblasts in many organs including the kidney. At the same time, the loss of pericytes within the perivascular compartment results in vulnerable capillaries which are prone to instability, pathological angiogenesis, and, ultimately, rarefaction. AIMS: This review will give an overview of pericyte-endothelial cell interactions, summarize the signaling pathways that have been identified to be involved in pericyte detachment from the vascular wall, and present pathological endothelial responses in the context of disease of the kidney. |Capillaries/cytology/physiology [MESH] |Cell Communication/physiology [MESH] |Cicatrix/pathology/physiopathology [MESH] |Endothelial Cells/*cytology/*physiology [MESH] |Fibrosis/pathology/physiopathology [MESH] |Humans [MESH] |Pericytes/*cytology/*physiology [MESH]The role of pericyte detachment in vascular rarefaction (epmc).pdf DeepDyve Pubget Overpricing