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10.1259/bjr.20130738 http://scihub22266oqcxt.onion/10.1259/bjr.20130738 C4453133!4453133 !25009948     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25009948 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25009948 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Br+J+Radiol 2014 ; 87 (1041 ): 20130738 Nephropedia Template TP {{tp|p=25009948 |t=2014. The role of inflammation in contrast-induced nephropathy |pdf=|usr=}}{{25009948 }} gab.com Text The role of inflammation in contrast-induced nephropathy JO: Br J Radiol http://www.kidney.de/pget.php?&tw=1&pmid=25009948 #FOAvip OBJECTIVE: Global incidence of contrast-induced nephropathy (CIN) is 2-5%, but a recent Kenyan study highlighted a local incidence of 12-14% without offering an explanation for the higher incidence. This study proposes that inflammatory states confer a higher relative risk for development of CIN. Our objective was to determine the risk of developing CIN given the presence of an inflammatory state in patients in Kenya. METHODS: Prospective cohort study of patients undergoing a contrast-enhanced CT (CECT) scan in a private university teaching hospital in Kenya and having no known risk factors for CIN. 423 patients were recruited and grouped into those without inflammation (unexposed) having serum C-reactive protein (CRP) levels ?5 mg dl(-1) and those with evidence of inflammation having CRP levels >5 mg dl(-1). Serum creatinine (SCr) was measured before the CECT and 48 h following the CECT with CIN diagnosed by an increase of >25% in the SCr from the baseline. Relative risk was determined and multiple logistic regression analysis performed on biophysical variables and contrast volume to assess their effect on development of CIN. RESULTS: Patients with high CRP levels had a relative risk of developing CIN of 2.16 compared with those with normal levels of CRP (p = 0.016). No statistically significant association was seen between biophysical variables or volume of contrast and development of CIN. CONCLUSION: Ongoing inflammation doubles the likelihood of development of CIN. ADVANCES IN KNOWLEDGE: This study highlights the importance of inflammation as a risk factor in the development of CIN. Twit Text FOAVip The role of inflammation in contrast-induced nephropathy ????Br J Radiol http://www.kidney.de/pget.php?&tw=1&pmid=25009948 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25009948 #FOAvip English Wikipedia <ref>{{Cite pmid|25009948 }}</ref> The role of inflammation in contrast-induced nephropathy #MMPMID25009948 Kwasa EA ; Vinayak S ; Armstrong R Br J Radiol 2014[Sep]; 87 (1041 ): 20130738 PMID25009948 show ga OBJECTIVE: Global incidence of contrast-induced nephropathy (CIN) is 2-5%, but a recent Kenyan study highlighted a local incidence of 12-14% without offering an explanation for the higher incidence. This study proposes that inflammatory states confer a higher relative risk for development of CIN. Our objective was to determine the risk of developing CIN given the presence of an inflammatory state in patients in Kenya. METHODS: Prospective cohort study of patients undergoing a contrast-enhanced CT (CECT) scan in a private university teaching hospital in Kenya and having no known risk factors for CIN. 423 patients were recruited and grouped into those without inflammation (unexposed) having serum C-reactive protein (CRP) levels ?5 mg dl(-1) and those with evidence of inflammation having CRP levels >5 mg dl(-1). Serum creatinine (SCr) was measured before the CECT and 48 h following the CECT with CIN diagnosed by an increase of >25% in the SCr from the baseline. Relative risk was determined and multiple logistic regression analysis performed on biophysical variables and contrast volume to assess their effect on development of CIN. RESULTS: Patients with high CRP levels had a relative risk of developing CIN of 2.16 compared with those with normal levels of CRP (p = 0.016). No statistically significant association was seen between biophysical variables or volume of contrast and development of CIN. CONCLUSION: Ongoing inflammation doubles the likelihood of development of CIN. ADVANCES IN KNOWLEDGE: This study highlights the importance of inflammation as a risk factor in the development of CIN. |Adult [MESH] |Aged [MESH] |C-Reactive Protein/analysis [MESH] |Contrast Media/*adverse effects [MESH] |Female [MESH] |Humans [MESH] |Incidence [MESH] |Inflammation/*physiopathology [MESH] |Kidney Diseases/*chemically induced/diagnostic imaging/*physiopathology [MESH] |Logistic Models [MESH] |Male [MESH] |Middle Aged [MESH] |Prospective Studies [MESH] |Radiography [MESH]The role of inflammation in contrast-induced nephropathy (epmc).pdf DeepDyve Pubget Overpricing