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10.1007/s11910-015-0534-4 http://scihub22266oqcxt.onion/10.1007/s11910-015-0534-4 C5489066!5489066 !25712420     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25712420 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Neurol+Neurosci+Rep 2015 ; 15 (4 ): 16 Nephropedia Template TP {{tp|p=25712420 |t=2015. The relationship between herpes zoster and stroke |pdf=|usr=}}{{25712420 }} gab.com Text The relationship between herpes zoster and stroke JO: Curr Neurol Neurosci Rep http://www.kidney.de/pget.php?&tw=1&pmid=25712420 #FOAvip Varicella zoster virus (VZV) infects >95 % of the world population. Typically, varicella (chickenpox) results from primary infection. The virus then becomes latent in ganglionic neurons along the entire neuraxis. In immunocompromised individuals, VZV reactivates and causes herpes zoster (shingles), pain, and rash in 1-2 dermatomes. Multiple case reports showed a link between stroke and zoster, and recent studies have emerged which reveal that VZV infection of the cerebral arteries directly causes pathological vascular remodeling and stroke (VZV vasculopathy). In the past few years, several large epidemiological studies in Taiwan, Denmark, and the U.K. demonstrated that zoster is a risk factor for stroke and that antiviral therapy may reduce this risk. Herein, the history, clinical features, and putative mechanisms of VZV vasculopathy, as well as recent epidemiological studies demonstrating that zoster increases the risk of stroke, are discussed. Twit Text FOAVip The relationship between herpes zoster and stroke ????Curr Neurol Neurosci Rep http://www.kidney.de/pget.php?&tw=1&pmid=25712420 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25712420 #FOAvip English Wikipedia <ref>{{Cite pmid|25712420 }}</ref> The relationship between herpes zoster and stroke #MMPMID25712420 Nagel MA ; Gilden D Curr Neurol Neurosci Rep 2015[Apr]; 15 (4 ): 16 PMID25712420 show ga Varicella zoster virus (VZV) infects >95 % of the world population. Typically, varicella (chickenpox) results from primary infection. The virus then becomes latent in ganglionic neurons along the entire neuraxis. In immunocompromised individuals, VZV reactivates and causes herpes zoster (shingles), pain, and rash in 1-2 dermatomes. Multiple case reports showed a link between stroke and zoster, and recent studies have emerged which reveal that VZV infection of the cerebral arteries directly causes pathological vascular remodeling and stroke (VZV vasculopathy). In the past few years, several large epidemiological studies in Taiwan, Denmark, and the U.K. demonstrated that zoster is a risk factor for stroke and that antiviral therapy may reduce this risk. Herein, the history, clinical features, and putative mechanisms of VZV vasculopathy, as well as recent epidemiological studies demonstrating that zoster increases the risk of stroke, are discussed. |Animals [MESH] |Herpes Zoster/*epidemiology/virology [MESH] |Herpesvirus 3, Human/*pathogenicity [MESH] |Humans [MESH]The relationship between herpes zoster and stroke (epmc).pdf DeepDyve Pubget Overpricing