Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1038/srep13538 http://scihub22266oqcxt.onion/10.1038/srep13538 C4642550!4642550!26337223     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26337223.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Sci+Rep 2015 ; 5 (ä): ä Nephropedia Template TP {{tp|p=26337223|t=2015. The motility-proliferation-metabolism interplay during metastatic invasion |pdf=|usr=}}{{26337223}} gab.com Text The motility-proliferation-metabolism interplay during metastatic invasion JO: Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=26337223 #FOAvip Metastasis is the major cause for cancer patients? death, and despite all the recent advances in cancer research it is still mostly incurable. Understanding the mechanisms that are involved in the migration of the cells in a complex environment is a key step towards successful anti-metastatic treatment. Using experimental data-based modeling, we focus on the fundamentals of metastatic invasion: motility, invasion, proliferation and metabolism, and study how they may be combined to maximize the cancer?s ability to metastasize. The modeled cells? performance is measured by the number of cells that succeed in migration in a maze, which mimics the extracellular environment. We show that co-existence of different cell clones in the tumor, as often found in experiments, optimizes the invasive ability in a frequently-changing environment. We study the role of metabolism and stimulation by growth factors, and show that metabolism plays a crucial role in the metastatic process and should therefore be targeted for successful treatment. Twit Text FOAVip The motility-proliferation-metabolism interplay during metastatic invasion ????Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=26337223 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26337223 #FOAvip English Wikipedia <ref>{{Cite pmid|26337223}}</ref> The motility-proliferation-metabolism interplay during metastatic invasion #MMPMID26337223 Hecht I; Natan S; Zaritsky A; Levine H; Tsarfaty I; Ben-Jacob E Sci Rep 2015[]; 5 (ä): ä PMID26337223show ga Metastasis is the major cause for cancer patients? death, and despite all the recent advances in cancer research it is still mostly incurable. Understanding the mechanisms that are involved in the migration of the cells in a complex environment is a key step towards successful anti-metastatic treatment. Using experimental data-based modeling, we focus on the fundamentals of metastatic invasion: motility, invasion, proliferation and metabolism, and study how they may be combined to maximize the cancer?s ability to metastasize. The modeled cells? performance is measured by the number of cells that succeed in migration in a maze, which mimics the extracellular environment. We show that co-existence of different cell clones in the tumor, as often found in experiments, optimizes the invasive ability in a frequently-changing environment. We study the role of metabolism and stimulation by growth factors, and show that metabolism plays a crucial role in the metastatic process and should therefore be targeted for successful treatment. äThe motility-proliferation-metabolism interplay during metastatic inva(epmc).pdf DeepDyve Pubget Overpricing