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10.1111/jcmm.12855 http://scihub22266oqcxt.onion/10.1111/jcmm.12855 C4988275!4988275 !27027405     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27027405 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27027405 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Cell+Mol+Med 2016 ; 20 (9 ): 1770-8 Nephropedia Template TP {{tp|p=27027405 |t=2016. The autophagy-lysosomal system in subarachnoid haemorrhage |pdf=|usr=}}{{27027405 }} gab.com Text The autophagy-lysosomal system in subarachnoid haemorrhage JO: J Cell Mol Med http://www.kidney.de/pget.php?&tw=1&pmid=27027405 #FOAvip The autophagy-lysosomal pathway is a self-catabolic process by which dysfunctional or unnecessary intracellular components are degraded by lysosomal enzymes. Proper function of this pathway is critical for maintaining cell homeostasis and survival. Subarachnoid haemorrhage (SAH) is one of the most devastating forms of stroke. Multiple pathogenic mechanisms, such as inflammation, apoptosis, and oxidative stress, are all responsible for brain injury and poor outcome after SAH. Most recently, accumulating evidence has demonstrated that the autophagy-lysosomal pathway plays a crucial role in the pathophysiological process after SAH. Appropriate activity of autophagy-lysosomal pathway acts as a pro-survival mechanism in SAH, while excessive self-digestion results in cell death after SAH. Consequently, in this review article, we will give an overview of the pathophysiological roles of autophagy-lysosomal pathway in the pathogenesis of SAH. And approaching the molecular mechanisms underlying this pathway in SAH pathology is anticipated, which may ultimately allow development of effective therapeutic strategies for SAH patients through regulating the autophagy-lysosomal machinery. Twit Text FOAVip The autophagy-lysosomal system in subarachnoid haemorrhage ????J Cell Mol Med http://www.kidney.de/pget.php?&tw=1&pmid=27027405 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27027405 #FOAvip English Wikipedia <ref>{{Cite pmid|27027405 }}</ref> The autophagy-lysosomal system in subarachnoid haemorrhage #MMPMID27027405 Wu H ; Niu H ; Wu C ; Li Y ; Wang K ; Zhang J ; Wang Y ; Yang S J Cell Mol Med 2016[Sep]; 20 (9 ): 1770-8 PMID27027405 show ga The autophagy-lysosomal pathway is a self-catabolic process by which dysfunctional or unnecessary intracellular components are degraded by lysosomal enzymes. Proper function of this pathway is critical for maintaining cell homeostasis and survival. Subarachnoid haemorrhage (SAH) is one of the most devastating forms of stroke. Multiple pathogenic mechanisms, such as inflammation, apoptosis, and oxidative stress, are all responsible for brain injury and poor outcome after SAH. Most recently, accumulating evidence has demonstrated that the autophagy-lysosomal pathway plays a crucial role in the pathophysiological process after SAH. Appropriate activity of autophagy-lysosomal pathway acts as a pro-survival mechanism in SAH, while excessive self-digestion results in cell death after SAH. Consequently, in this review article, we will give an overview of the pathophysiological roles of autophagy-lysosomal pathway in the pathogenesis of SAH. And approaching the molecular mechanisms underlying this pathway in SAH pathology is anticipated, which may ultimately allow development of effective therapeutic strategies for SAH patients through regulating the autophagy-lysosomal machinery. |*Autophagy [MESH] |Animals [MESH] |Brain Injuries/complications/pathology [MESH] |Cathepsins/metabolism [MESH] |Humans [MESH] |Lysosomes/*metabolism [MESH] |Models, Biological [MESH]The autophagy-lysosomal system in subarachnoid haemorrhage (epmc).pdf DeepDyve Pubget Overpricing