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10.3390/ijms19020562 http://scihub22266oqcxt.onion/10.3390/ijms19020562 C5855784!5855784 !29438305     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29438305 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Int+J+Mol+Sci 2018 ; 19 (2 ): ä Nephropedia Template TP {{tp|p=29438305 |t=2018. The Crosstalk between Nrf2 and Inflammasomes |pdf=|usr=}}{{29438305 }} gab.com Text The Crosstalk between Nrf2 and Inflammasomes JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=29438305 #FOAvip The Nrf2 (nuclear factor E2-related factor or nuclear factor (erythroid-derived 2)-like 2) transcription factor is a key player in cytoprotection and activated in stress conditions caused by reactive oxygen species (ROS) or electrophiles. Inflammasomes represent central regulators of inflammation. Upon detection of various stress factors, assembly of the inflamasome protein complex results in activation and secretion of proinflammatory cytokines. In addition, inflammasome activation causes pyroptosis, a lytic form of cell death, which supports inflammation. There is growing evidence of a crosstalk between the Nrf2 and inflammasome pathways at different levels. For example, Nrf2 activating compounds inhibit inflammasomes and consequently inflammation. This review summarizes what is known about the complex and predominantly antagonistic relationship of both stress-activated pathways. Twit Text FOAVip The Crosstalk between Nrf2 and Inflammasomes ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=29438305 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29438305 #FOAvip English Wikipedia <ref>{{Cite pmid|29438305 }}</ref> The Crosstalk between Nrf2 and Inflammasomes #MMPMID29438305 Hennig P ; Garstkiewicz M ; Grossi S ; Di Filippo M ; French LE ; Beer HD Int J Mol Sci 2018[Feb]; 19 (2 ): ä PMID29438305 show ga The Nrf2 (nuclear factor E2-related factor or nuclear factor (erythroid-derived 2)-like 2) transcription factor is a key player in cytoprotection and activated in stress conditions caused by reactive oxygen species (ROS) or electrophiles. Inflammasomes represent central regulators of inflammation. Upon detection of various stress factors, assembly of the inflamasome protein complex results in activation and secretion of proinflammatory cytokines. In addition, inflammasome activation causes pyroptosis, a lytic form of cell death, which supports inflammation. There is growing evidence of a crosstalk between the Nrf2 and inflammasome pathways at different levels. For example, Nrf2 activating compounds inhibit inflammasomes and consequently inflammation. This review summarizes what is known about the complex and predominantly antagonistic relationship of both stress-activated pathways. |Animals [MESH] |Humans [MESH] |Inflammasomes/genetics/*metabolism [MESH] |NF-E2-Related Factor 2/genetics/*metabolism [MESH]The Crosstalk between Nrf2 and Inflammasomes (epmc).pdf DeepDyve Pubget Overpricing