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10.1016/j.molcel.2016.05.013 http://scihub22266oqcxt.onion/10.1016/j.molcel.2016.05.013 C4917370!4917370!27259204     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27259204.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mol+Cell 2016 ; 62 (5): 728-44 Nephropedia Template TP {{tp|p=27259204|t=2016. The Aging Epigenome |pdf=|usr=}}{{27259204}} gab.com Text The Aging Epigenome JO: Mol Cell http://www.kidney.de/pget.php?&tw=1&pmid=27259204 #FOAvip During aging, the mechanisms that normally maintain health and stress resistance strikingly decline, resulting in decrepitude, frailty, and ultimately death. Exactly when and how this decline occurs is unknown. Changes in transcriptional networks and chromatin state lie at the heart of age-dependent decline. These epigenomic changes are not only observed during aging but also profoundly affect cellular function and stress resistance, thereby contributing to the progression of aging. We propose that the dysregulation of transcriptional and chromatin networks is a crucial component of aging. Understanding age-dependent epigenomic changes will yield key insights into how aging begins and progresses and should lead to the development of new therapeutics that delay or even reverse aging and age-related diseases. Twit Text FOAVip The Aging Epigenome ????Mol Cell http://www.kidney.de/pget.php?&tw=1&pmid=27259204 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27259204 #FOAvip English Wikipedia <ref>{{Cite pmid|27259204}}</ref> The Aging Epigenome #MMPMID27259204 Booth LN; Brunet A Mol Cell 2016[Jun]; 62 (5): 728-44 PMID27259204show ga During aging, the mechanisms that normally maintain health and stress resistance strikingly decline, resulting in decrepitude, frailty, and ultimately death. Exactly when and how this decline occurs is unknown. Changes in transcriptional networks and chromatin state lie at the heart of age-dependent decline. These epigenomic changes are not only observed during aging but also profoundly affect cellular function and stress resistance, thereby contributing to the progression of aging. We propose that the dysregulation of transcriptional and chromatin networks is a crucial component of aging. Understanding age-dependent epigenomic changes will yield key insights into how aging begins and progresses and should lead to the development of new therapeutics that delay or even reverse aging and age-related diseases. äThe Aging Epigenome (epmc).pdf DeepDyve Pubget Overpricing