Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.semnephrol.2016.01.003 http://scihub22266oqcxt.onion/10.1016/j.semnephrol.2016.01.003 C5439503!5439503 !27085736     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27085736 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Semin+Nephrol 2016 ; 36 (1 ): 62-70 Nephropedia Template TP {{tp|p=27085736 |t=2016. Targeting Iron Homeostasis in Acute Kidney Injury |pdf=|usr=}}{{27085736 }} gab.com Text Targeting Iron Homeostasis in Acute Kidney Injury JO: Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=27085736 #FOAvip Iron is an essential metal involved in several major cellular processes required to maintain life. Because of iron's ability to cause oxidative damage, its transport, metabolism, and storage is strictly controlled in the body, especially in the small intestine, liver, and kidney. Iron plays a major role in acute kidney injury and has been a target for therapeutic intervention. However, the therapies that have been effective in animal models of acute kidney injury have not been successful in human beings. Targeting iron trafficking via ferritin, ferroportin, or hepcidin may offer new insights. This review focuses on the biology of iron, particularly in the kidney, and its implications in acute kidney injury. Twit Text FOAVip Targeting Iron Homeostasis in Acute Kidney Injury ????Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=27085736 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27085736 #FOAvip English Wikipedia <ref>{{Cite pmid|27085736 }}</ref> Targeting Iron Homeostasis in Acute Kidney Injury #MMPMID27085736 Walker VJ ; Agarwal A Semin Nephrol 2016[Jan]; 36 (1 ): 62-70 PMID27085736 show ga Iron is an essential metal involved in several major cellular processes required to maintain life. Because of iron's ability to cause oxidative damage, its transport, metabolism, and storage is strictly controlled in the body, especially in the small intestine, liver, and kidney. Iron plays a major role in acute kidney injury and has been a target for therapeutic intervention. However, the therapies that have been effective in animal models of acute kidney injury have not been successful in human beings. Targeting iron trafficking via ferritin, ferroportin, or hepcidin may offer new insights. This review focuses on the biology of iron, particularly in the kidney, and its implications in acute kidney injury. |Acute Kidney Injury/*drug therapy/*metabolism [MESH] |Ferritins/*blood [MESH] |Hepcidins/*therapeutic use [MESH] |Homeostasis/*physiology [MESH] |Humans [MESH]Targeting Iron Homeostasis in Acute Kidney Injury (epmc).pdf DeepDyve Pubget Overpricing