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10.7150/jca.14031 http://scihub22266oqcxt.onion/10.7150/jca.14031 C4911874!4911874 !27326250     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27326250 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Cancer 2016 ; 7 (9 ): 1074-80 Nephropedia Template TP {{tp|p=27326250 |t=2016. TWIST1 and BMI1 in Cancer Metastasis and Chemoresistance |pdf=|usr=}}{{27326250 }} gab.com Text TWIST1 and BMI1 in Cancer Metastasis and Chemoresistance JO: J Cancer http://www.kidney.de/pget.php?&tw=1&pmid=27326250 #FOAvip Purpose Increasing evidences revealed that cancer cells with the characteristics of epithelial-mesenchymal transition (EMT) or cancer stem cells (CSC) have high ability of progression, invasion, metastasis and chemoresistance. TWIST1 and BMI1 are crucial transcription factors required for EMT and CSC. Both TWIST1 and BMI1 are up-regulated in various cancers and have a positive correlation with poor prognosis. Although recent results showed that the two molecules function in promoting cancer metastasis and chemoresistance respectively, the correlation of TWIST1 and BMI1 is not well understood. Methods In this review, we summarize recent advance in cancer research focus on TWIST1 and BMI1 in cancer metastasis and chemoresistance, and emphasize the possible link between EMT and CSC. Results Further investigation of TWIST1 and BMI1 cooperately promote CSC proliferation due to EMT-associated effect will help to understand the mechanism of tumor cells metastasis and chemoresistance. Conclusions TWIST1 and BMI1 in cancer cells will be effective targets for treating chemoresistant metastatic lesions. Twit Text FOAVip TWIST1 and BMI1 in Cancer Metastasis and Chemoresistance ????J Cancer http://www.kidney.de/pget.php?&tw=1&pmid=27326250 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27326250 #FOAvip English Wikipedia <ref>{{Cite pmid|27326250 }}</ref> TWIST1 and BMI1 in Cancer Metastasis and Chemoresistance #MMPMID27326250 Ren H ; Du P ; Ge Z ; Jin Y ; Ding D ; Liu X ; Zou Q J Cancer 2016[]; 7 (9 ): 1074-80 PMID27326250 show ga Purpose Increasing evidences revealed that cancer cells with the characteristics of epithelial-mesenchymal transition (EMT) or cancer stem cells (CSC) have high ability of progression, invasion, metastasis and chemoresistance. TWIST1 and BMI1 are crucial transcription factors required for EMT and CSC. Both TWIST1 and BMI1 are up-regulated in various cancers and have a positive correlation with poor prognosis. Although recent results showed that the two molecules function in promoting cancer metastasis and chemoresistance respectively, the correlation of TWIST1 and BMI1 is not well understood. Methods In this review, we summarize recent advance in cancer research focus on TWIST1 and BMI1 in cancer metastasis and chemoresistance, and emphasize the possible link between EMT and CSC. Results Further investigation of TWIST1 and BMI1 cooperately promote CSC proliferation due to EMT-associated effect will help to understand the mechanism of tumor cells metastasis and chemoresistance. Conclusions TWIST1 and BMI1 in cancer cells will be effective targets for treating chemoresistant metastatic lesions. ?TWIST1 and BMI1 in Cancer Metastasis and Chemoresistance (epmc).pdf DeepDyve Pubget Overpricing