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10.15252/embj.201489988 http://scihub22266oqcxt.onion/10.15252/embj.201489988 C4282569!4282569 !25266476     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25266476 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25266476 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       EMBO+J 2014 ; 33 (22 ): 2601-3 Nephropedia Template TP {{tp|p=25266476 |t=2014. Splicing together sister chromatids |pdf=|usr=}}{{25266476 }} gab.com Text Splicing together sister chromatids JO: EMBO J http://www.kidney.de/pget.php?&tw=1&pmid=25266476 #FOAvip Splicing of pre?mRNAs is a necessary step for expression of the majority of genes in higher eukaryotes, and its regulation through alternative splice site selection shapes their proteomes. Defects in multiple splicing factors result in aberrant mitotic progression, although the molecular basis for this observation has remained elusive. Recent papers in The EMBO Journal and EMBO Reports reveal that expression of sororin, a critical regulator that stabilizes cohesin rings in sister chromatids, is exquisitely sensitive to defects in the splicing machinery, thus explaining the striking link between spliceosome function and chromosome segregation. Twit Text FOAVip Splicing together sister chromatids ????EMBO J http://www.kidney.de/pget.php?&tw=1&pmid=25266476 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25266476 #FOAvip English Wikipedia <ref>{{Cite pmid|25266476 }}</ref> Splicing together sister chromatids #MMPMID25266476 Valcárcel J ; Malumbres M EMBO J 2014[Nov]; 33 (22 ): 2601-3 PMID25266476 show ga Splicing of pre?mRNAs is a necessary step for expression of the majority of genes in higher eukaryotes, and its regulation through alternative splice site selection shapes their proteomes. Defects in multiple splicing factors result in aberrant mitotic progression, although the molecular basis for this observation has remained elusive. Recent papers in The EMBO Journal and EMBO Reports reveal that expression of sororin, a critical regulator that stabilizes cohesin rings in sister chromatids, is exquisitely sensitive to defects in the splicing machinery, thus explaining the striking link between spliceosome function and chromosome segregation. |*Leukemia, Lymphocytic, Chronic, B-Cell [MESH] |*Mutation [MESH] |*Neoplasm Proteins [MESH] |*Phosphoproteins [MESH] |*RNA, Neoplasm [MESH] |*Ribonucleoprotein, U2 Small Nuclear [MESH] |*Sister Chromatid Exchange [MESH] |Adaptor Proteins, Signal Transducing/*metabolism [MESH] |Cell Cycle Proteins/*metabolism [MESH] |Chromatids/*metabolism [MESH] |Cytoskeletal Proteins/*metabolism [MESH] |Humans [MESH] |Nuclear Receptor Coactivators/*metabolism [MESH] |RNA Precursors/*metabolism [MESH] |RNA Splicing Factors [MESH]Splicing together sister chromatids (epmc).pdf DeepDyve Pubget Overpricing