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10.1016/j.it.2016.04.007 http://scihub22266oqcxt.onion/10.1016/j.it.2016.04.007 C4931795!4931795!27178391     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27178391.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Trends+Immunol 2016 ; 37 (7): 440-50 Nephropedia Template TP {{tp|p=27178391|t=2016. Skin Immunity to Candida albicans |pdf=|usr=}}{{27178391}} gab.com Text Skin Immunity to Candida albicans JO: Trends Immunol http://www.kidney.de/pget.php?&tw=1&pmid=27178391 #FOAvip Candida albicans is a dimorphic commensal fungus that colonizes healthy human skin, mucosa and reproductive tract. C. albicans is also a predominant opportunistic fungal pathogen, leading to disease manifestations such as disseminated candidiasis and chronic mucocutaneous candidiasis (CMC). The differing host susceptibilities to the sites of C. albicans infection have revealed tissue compartmentalization with tailoring of immune responses based on site of infection. Furthermore, extensive studies of host genetics in rare cases of CMC have identified conserved genetic pathways involved in the immune recognition and response to the extracellular pathogens. Herein, we focus on the human and mouse skin as a site of C. albicans infection and review the established and newly discovered insights into those cellular pathways that promote cutaneous anti-fungal immunity. Twit Text FOAVip Skin Immunity to Candida albicans ????Trends Immunol http://www.kidney.de/pget.php?&tw=1&pmid=27178391 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27178391 #FOAvip English Wikipedia <ref>{{Cite pmid|27178391}}</ref> Skin Immunity to Candida albicans #MMPMID27178391 Kashem SW; Kaplan DH Trends Immunol 2016[Jul]; 37 (7): 440-50 PMID27178391show ga Candida albicans is a dimorphic commensal fungus that colonizes healthy human skin, mucosa and reproductive tract. C. albicans is also a predominant opportunistic fungal pathogen, leading to disease manifestations such as disseminated candidiasis and chronic mucocutaneous candidiasis (CMC). The differing host susceptibilities to the sites of C. albicans infection have revealed tissue compartmentalization with tailoring of immune responses based on site of infection. Furthermore, extensive studies of host genetics in rare cases of CMC have identified conserved genetic pathways involved in the immune recognition and response to the extracellular pathogens. Herein, we focus on the human and mouse skin as a site of C. albicans infection and review the established and newly discovered insights into those cellular pathways that promote cutaneous anti-fungal immunity. äSkin Immunity to Candida albicans (epmc).pdf DeepDyve Pubget Overpricing