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10.1016/j.it.2016.04.007 http://scihub22266oqcxt.onion/10.1016/j.it.2016.04.007 C4931795!4931795 !27178391     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27178391 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Trends+Immunol 2016 ; 37 (7 ): 440-450 Nephropedia Template TP {{tp|p=27178391 |t=2016. Skin Immunity to Candida albicans |pdf=|usr=}}{{27178391 }} gab.com Text Skin Immunity to Candida albicans JO: Trends Immunol http://www.kidney.de/pget.php?&tw=1&pmid=27178391 #FOAvip Candida albicans is a dimorphic commensal fungus that colonizes healthy human skin, mucosa, and the reproductive tract. C. albicans is also a predominantly opportunistic fungal pathogen, leading to disease manifestations such as disseminated candidiasis and chronic mucocutaneous candidiasis (CMC). The differing host susceptibilities for the sites of C. albicans infection have revealed tissue compartmentalization with tailoring of immune responses based on the site of infection. Furthermore, extensive studies of host genetics in rare cases of CMC have identified conserved genetic pathways involved in immune recognition and the response to the extracellular pathogen. We focus here on human and mouse skin as a site of C. albicans infection, and we review established and newly discovered insights into the cellular pathways that promote cutaneous antifungal immunity. Twit Text FOAVip Skin Immunity to Candida albicans ????Trends Immunol http://www.kidney.de/pget.php?&tw=1&pmid=27178391 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27178391 #FOAvip English Wikipedia <ref>{{Cite pmid|27178391 }}</ref> Skin Immunity to Candida albicans #MMPMID27178391 Kashem SW ; Kaplan DH Trends Immunol 2016[Jul]; 37 (7 ): 440-450 PMID27178391 show ga Candida albicans is a dimorphic commensal fungus that colonizes healthy human skin, mucosa, and the reproductive tract. C. albicans is also a predominantly opportunistic fungal pathogen, leading to disease manifestations such as disseminated candidiasis and chronic mucocutaneous candidiasis (CMC). The differing host susceptibilities for the sites of C. albicans infection have revealed tissue compartmentalization with tailoring of immune responses based on the site of infection. Furthermore, extensive studies of host genetics in rare cases of CMC have identified conserved genetic pathways involved in immune recognition and the response to the extracellular pathogen. We focus here on human and mouse skin as a site of C. albicans infection, and we review established and newly discovered insights into the cellular pathways that promote cutaneous antifungal immunity. |*Immunity [MESH] |Animals [MESH] |Candida albicans/*immunology [MESH] |Candidiasis/*immunology [MESH] |Genetic Predisposition to Disease [MESH] |Humans [MESH] |Mice [MESH] |Opportunistic Infections/*immunology [MESH] |Organ Specificity [MESH]Skin Immunity to Candida albicans (epmc).pdf DeepDyve Pubget Overpricing