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10.1007/s10555-014-9518-0 http://scihub22266oqcxt.onion/10.1007/s10555-014-9518-0 C4425804!4425804 !25394532     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25394532 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Cancer+Metastasis+Rev 2014 ; 33 (4 ): 921-8 Nephropedia Template TP {{tp|p=25394532 |t=2014. Role of MTA2 in human cancer |pdf=|usr=}}{{25394532 }} gab.com Text Role of MTA2 in human cancer JO: Cancer Metastasis Rev http://www.kidney.de/pget.php?&tw=1&pmid=25394532 #FOAvip Metastasis is the ultimate cause of death for most cancer patients. While many mechanisms have been delineated for regulation of growth and tumor initiation of the primary tumor, very little is known about the process of metastasis. Metastasis requires dynamic alteration of cellular processes in order for cells to disseminate from the primary tumor to distant sites. These alterations often involve dramatic changes in the regulation of cytoskeletal and cell-environment interactions. Furthermore, controlled refinement of these interactions requires feedback to regulatory networks in the nucleus. MTA2 is a member of the metastasis tumor-associated family of transcriptional regulators and is a central component of the nucleosome remodeling and histone deacetylation complex. MTA2 acts as a central hub for cytoskeletal organization and transcription and provides a link between nuclear and cytoskeletal organization. We will focus on MTA2 in this chapter, especially its role in breast cancer metastasis. Twit Text FOAVip Role of MTA2 in human cancer ????Cancer Metastasis Rev http://www.kidney.de/pget.php?&tw=1&pmid=25394532 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25394532 #FOAvip English Wikipedia <ref>{{Cite pmid|25394532 }}</ref> Role of MTA2 in human cancer #MMPMID25394532 Covington KR ; Fuqua SA Cancer Metastasis Rev 2014[Dec]; 33 (4 ): 921-8 PMID25394532 show ga Metastasis is the ultimate cause of death for most cancer patients. While many mechanisms have been delineated for regulation of growth and tumor initiation of the primary tumor, very little is known about the process of metastasis. Metastasis requires dynamic alteration of cellular processes in order for cells to disseminate from the primary tumor to distant sites. These alterations often involve dramatic changes in the regulation of cytoskeletal and cell-environment interactions. Furthermore, controlled refinement of these interactions requires feedback to regulatory networks in the nucleus. MTA2 is a member of the metastasis tumor-associated family of transcriptional regulators and is a central component of the nucleosome remodeling and histone deacetylation complex. MTA2 acts as a central hub for cytoskeletal organization and transcription and provides a link between nuclear and cytoskeletal organization. We will focus on MTA2 in this chapter, especially its role in breast cancer metastasis. |Breast Neoplasms/*genetics/pathology [MESH] |Cytoskeleton/genetics [MESH] |Female [MESH] |Gene Expression Regulation, Neoplastic [MESH] |Histone Deacetylases/biosynthesis/*genetics [MESH] |Humans [MESH] |Mi-2 Nucleosome Remodeling and Deacetylase Complex/genetics [MESH] |Neoplasm Metastasis [MESH] |Neoplasms/*genetics/pathology [MESH]Role of MTA2 in human cancer (epmc).pdf DeepDyve Pubget Overpricing