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10.1155/2015/186908 http://scihub22266oqcxt.onion/10.1155/2015/186908 C4443900!4443900 !26064416     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26064416 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Oxid+Med+Cell+Longev 2015 ; 2015 (ä): 186908 Nephropedia Template TP {{tp|p=26064416 |t=2015. Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury |pdf=|usr=}}{{26064416 }} gab.com Text Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury JO: Oxid Med Cell Longev http://www.kidney.de/pget.php?&tw=1&pmid=26064416 #FOAvip Ischemia-reperfusion (I/R) injury is one of the major causes of high morbidity, disability, and mortality in the world. I/R injury remains a complicated and unresolved situation in clinical practice, especially in the field of solid organ transplantation. Hydrogen sulfide (H2S) is the third gaseous signaling molecule and plays a broad range of physiological and pathophysiological roles in mammals. H2S could protect against I/R injury in many organs and tissues, such as heart, liver, kidney, brain, intestine, stomach, hind-limb, lung, and retina. The goal of this review is to highlight recent findings regarding the role of H2S in I/R injury. In this review, we present the production and metabolism of H2S and further discuss the effect and mechanism of H2S in I/R injury. Twit Text FOAVip Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury ????Oxid Med Cell Longev http://www.kidney.de/pget.php?&tw=1&pmid=26064416 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26064416 #FOAvip English Wikipedia <ref>{{Cite pmid|26064416 }}</ref> Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury #MMPMID26064416 Wu D ; Wang J ; Li H ; Xue M ; Ji A ; Li Y Oxid Med Cell Longev 2015[]; 2015 (ä): 186908 PMID26064416 show ga Ischemia-reperfusion (I/R) injury is one of the major causes of high morbidity, disability, and mortality in the world. I/R injury remains a complicated and unresolved situation in clinical practice, especially in the field of solid organ transplantation. Hydrogen sulfide (H2S) is the third gaseous signaling molecule and plays a broad range of physiological and pathophysiological roles in mammals. H2S could protect against I/R injury in many organs and tissues, such as heart, liver, kidney, brain, intestine, stomach, hind-limb, lung, and retina. The goal of this review is to highlight recent findings regarding the role of H2S in I/R injury. In this review, we present the production and metabolism of H2S and further discuss the effect and mechanism of H2S in I/R injury. |Animals [MESH] |Humans [MESH] |Hydrogen Sulfide/*metabolism [MESH] |Myocardial Reperfusion Injury/*metabolism/pathology [MESH]Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury (epmc).pdf DeepDyve Pubget Overpricing