Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28348661
&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests
in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215
Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28348661
.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117 Cardiol+Res
2011 ; 2
(1
): 48-49
Nephropedia Template TP
gab.com Text
Twit Text FOAVip
Twit Text #
English Wikipedia
Right Ventricle Failure in Sepsis: A Case Report
#MMPMID28348661
Lakshmanadoss U
; Levitan BM
; Hsi DH
Cardiol Res
2011[Feb]; 2
(1
): 48-49
PMID28348661
show ga
Sepsis could produce myocardial depression and typically it affects the left
ventricle (LV). Sepsis could also affect right ventricle (RV), in addition to the
interdependence with LV. RV pressure may be elevated secondary to pulmonary
vasoconstriction, leading to RV dysfunction. Unlike LV, RV is poorly prepared to
compensate for acute overload. Aggressive volume replacement may be vital to
maintain RV function, but excess hydration can cause RV dilation, decreased LV
diastolic filling, and reduced cardiac output. In patients having signs of
inadequate cardiac output even after initial volume resuscitation, RV function
should be assessed with echocardiogram. If RV dysfunction is noted, then fluid
therapy should be guided by CVP measurements. If cardiac output increases with
increasing CVP, maintaining higher filling pressures on the right side is
indicated. On the other hand, increasing CVP with worsening of the cardiac output
could worsen the RV dysfunction. In addition to the fluid management, treatment
of other reversible causes like acidosis and hypoxia is also a key.