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10.1016/j.siny.2017.01.001 http://scihub22266oqcxt.onion/10.1016/j.siny.2017.01.001 C5387761!5387761 !28161315     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28161315 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28161315 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Semin+Fetal+Neonatal+Med 2017 ; 22 (2 ): 58-66 Nephropedia Template TP {{tp|p=28161315 |t=2017. Renal development in the fetus and premature infant |pdf=|usr=}}{{28161315 }} gab.com Text Renal development in the fetus and premature infant JO: Semin Fetal Neonatal Med http://www.kidney.de/pget.php?&tw=1&pmid=28161315 #FOAvip Congenital abnormalities of the kidney and urinary tract (CAKUT) are one of the leading congenital defects to be identified on prenatal ultrasound. CAKUT represent a broad spectrum of abnormalities, from transient hydronephrosis to severe bilateral renal agenesis. CAKUT are a major contributor to chronic and end stage kidney disease (CKD/ESKD) in children. Prenatal imaging is useful to identify CAKUT, but will not detect all defects. Both genetic abnormalities and the fetal environment contribute to CAKUT. Monogenic gene mutations identified in human CAKUT have advanced our understanding of molecular mechanisms of renal development. Low nephron number and solitary kidneys are associated with increased risk of adult onset CKD and ESKD. Premature and low birth weight infants represent a high risk population for low nephron number. Additional research is needed to identify biomarkers and appropriate follow-up of premature and low birth weight infants into adulthood. Twit Text FOAVip Renal development in the fetus and premature infant ????Semin Fetal Neonatal Med http://www.kidney.de/pget.php?&tw=1&pmid=28161315 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28161315 #FOAvip English Wikipedia <ref>{{Cite pmid|28161315 }}</ref> Renal development in the fetus and premature infant #MMPMID28161315 Rosenblum S ; Pal A ; Reidy K Semin Fetal Neonatal Med 2017[Apr]; 22 (2 ): 58-66 PMID28161315 show ga Congenital abnormalities of the kidney and urinary tract (CAKUT) are one of the leading congenital defects to be identified on prenatal ultrasound. CAKUT represent a broad spectrum of abnormalities, from transient hydronephrosis to severe bilateral renal agenesis. CAKUT are a major contributor to chronic and end stage kidney disease (CKD/ESKD) in children. Prenatal imaging is useful to identify CAKUT, but will not detect all defects. Both genetic abnormalities and the fetal environment contribute to CAKUT. Monogenic gene mutations identified in human CAKUT have advanced our understanding of molecular mechanisms of renal development. Low nephron number and solitary kidneys are associated with increased risk of adult onset CKD and ESKD. Premature and low birth weight infants represent a high risk population for low nephron number. Additional research is needed to identify biomarkers and appropriate follow-up of premature and low birth weight infants into adulthood. |*Ultrasonography [MESH] |Humans [MESH] |Infant, Premature [MESH] |Infant, Premature, Diseases/*diagnostic imaging [MESH] |Kidney Diseases/*diagnostic imaging [MESH] |Kidney/diagnostic imaging/*embryology [MESH]Renal development in the fetus and premature infant (epmc).pdf DeepDyve Pubget Overpricing