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10.1097/MNH.0000000000000202 http://scihub22266oqcxt.onion/10.1097/MNH.0000000000000202 C4768864!4768864 !26741885     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26741885 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26741885 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Opin+Nephrol+Hypertens 2016 ; 25 (2 ): 144-51 Nephropedia Template TP {{tp|p=26741885 |t=2016. Renal artery stenosis: if and when to intervene |pdf=|usr=}}{{26741885 }} gab.com Text Renal artery stenosis: if and when to intervene JO: Curr Opin Nephrol Hypertens http://www.kidney.de/pget.php?&tw=1&pmid=26741885 #FOAvip PURPOSE OF REVIEW: Atherosclerotic renovascular disease remains highly prevalent and presents an array of clinical syndromes. Recent prospective trials have dampened enthusiasm for revascularization generally, but clinicians recognize the need to identify patients likely to benefit from vascular intervention. RECENT FINDINGS: This article highlights the inflammatory nature of vascular occlusive disease and the limits of the kidney to adapt to reduced blood flow. Although moderate reductions can be tolerated, severe impairment of renal perfusion leads to tissue hypoxia and activates inflammatory injury within the kidney. Hence, assessment of kidney viability and potential tools to modify mitochondrial and inflammatory damage may be important to identify patients for whom clinical intervention should be undertaken. SUMMARY: Clinicians must recognize clinical syndromes that identify 'high-risk' groups and apply revascularization in those likely to benefit. Future efforts to protect the kidney (e.g., mitochondrial protection) or cell-based therapy may amplify clinical recovery when combined with restoring renal blood flow. Twit Text FOAVip Renal artery stenosis: if and when to intervene ????Curr Opin Nephrol Hypertens http://www.kidney.de/pget.php?&tw=1&pmid=26741885 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26741885 #FOAvip English Wikipedia <ref>{{Cite pmid|26741885 }}</ref> Renal artery stenosis: if and when to intervene #MMPMID26741885 Textor SC ; McKusick MM Curr Opin Nephrol Hypertens 2016[Mar]; 25 (2 ): 144-51 PMID26741885 show ga PURPOSE OF REVIEW: Atherosclerotic renovascular disease remains highly prevalent and presents an array of clinical syndromes. Recent prospective trials have dampened enthusiasm for revascularization generally, but clinicians recognize the need to identify patients likely to benefit from vascular intervention. RECENT FINDINGS: This article highlights the inflammatory nature of vascular occlusive disease and the limits of the kidney to adapt to reduced blood flow. Although moderate reductions can be tolerated, severe impairment of renal perfusion leads to tissue hypoxia and activates inflammatory injury within the kidney. Hence, assessment of kidney viability and potential tools to modify mitochondrial and inflammatory damage may be important to identify patients for whom clinical intervention should be undertaken. SUMMARY: Clinicians must recognize clinical syndromes that identify 'high-risk' groups and apply revascularization in those likely to benefit. Future efforts to protect the kidney (e.g., mitochondrial protection) or cell-based therapy may amplify clinical recovery when combined with restoring renal blood flow. |Adaptation, Physiological [MESH] |Angioplasty [MESH] |Animals [MESH] |Atherosclerosis/*complications [MESH] |Humans [MESH] |Inflammation/*complications [MESH] |Kidney/*blood supply/physiopathology [MESH] |Patient Selection [MESH]Renal artery stenosis: if and when to intervene (epmc).pdf DeepDyve Pubget Overpricing