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10.1186/s12950-017-0159-2 http://scihub22266oqcxt.onion/10.1186/s12950-017-0159-2 C5463399!5463399 !28596706     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28596706 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Inflamm+(Lond) 2017 ; 14 (ä): 11 Nephropedia Template TP {{tp|p=28596706 |t=2017. Regulating STING in health and disease |pdf=|usr=}}{{28596706 }} gab.com Text Regulating STING in health and disease JO: J Inflamm (Lond) http://www.kidney.de/pget.php?&tw=1&pmid=28596706 #FOAvip The presence of cytosolic double-stranded DNA molecules can trigger multiple innate immune signalling pathways which converge on the activation of an ER-resident innate immune adaptor named "STimulator of INterferon Genes (STING)". STING has been found to mediate type I interferon response downstream of cyclic dinucleotides and a number of DNA and RNA inducing signalling pathway. In addition to its physiological function, a rapidly increasing body of literature highlights the role for STING in human disease where variants of the STING proteins, as well as dysregulated STING signalling, have been implicated in a number of inflammatory diseases. This review will summarise the recent structural and functional findings of STING, and discuss how STING research has promoted the development of novel therapeutic approaches and experimental tools to improve treatment of tumour and autoimmune diseases. Twit Text FOAVip Regulating STING in health and disease ????J Inflamm (Lond) http://www.kidney.de/pget.php?&tw=1&pmid=28596706 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28596706 #FOAvip English Wikipedia <ref>{{Cite pmid|28596706 }}</ref> Regulating STING in health and disease #MMPMID28596706 Li Y ; Wilson HL ; Kiss-Toth E J Inflamm (Lond) 2017[]; 14 (ä): 11 PMID28596706 show ga The presence of cytosolic double-stranded DNA molecules can trigger multiple innate immune signalling pathways which converge on the activation of an ER-resident innate immune adaptor named "STimulator of INterferon Genes (STING)". STING has been found to mediate type I interferon response downstream of cyclic dinucleotides and a number of DNA and RNA inducing signalling pathway. In addition to its physiological function, a rapidly increasing body of literature highlights the role for STING in human disease where variants of the STING proteins, as well as dysregulated STING signalling, have been implicated in a number of inflammatory diseases. This review will summarise the recent structural and functional findings of STING, and discuss how STING research has promoted the development of novel therapeutic approaches and experimental tools to improve treatment of tumour and autoimmune diseases. äRegulating STING in health and disease (epmc).pdf DeepDyve Pubget Overpricing