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10.3892/ol.2016.5200 http://scihub22266oqcxt.onion/10.3892/ol.2016.5200 C5104145!5104145 !27900053     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27900053 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27900053 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Oncol+Lett 2016 ; 12 (5 ): 3679-3682 Nephropedia Template TP {{tp|p=27900053 |t=2016. Receptor tyrosine kinases in carcinogenesis |pdf=|usr=}}{{27900053 }} gab.com Text Receptor tyrosine kinases in carcinogenesis JO: Oncol Lett http://www.kidney.de/pget.php?&tw=1&pmid=27900053 #FOAvip Receptor tyrosine kinases (RTKs) are cell surface glycoproteins with enzymatic activity involved in the regulation of various important functions. In all-important physiological functions including differentiation, cell-cell interactions, survival, proliferation, metabolism, migration and signaling these receptors are the key players of regulation. Additionally, mutations of RTKs or their overexpression have been described in many human cancers and are being explored as a novel avenue for a new therapeutic approach. Some of the deregulated RTKs observed to be significantly affected in cancers included vascular endothelial growth factor receptor, epidermal growth factor receptor, fibroblast growth factor receptor, RTK-like orphan receptor 1 (ROR1) and the platelet-derived growth factor receptor. These deregulated RTKs offer attractive possibilities for the new anticancer therapeutic approach involving specific targeting by monoclonal antibodies as well as kinase. The present review aimed to highlight recent perspectives of RTK ROR1 in cancer. Twit Text FOAVip Receptor tyrosine kinases in carcinogenesis ????Oncol Lett http://www.kidney.de/pget.php?&tw=1&pmid=27900053 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27900053 #FOAvip English Wikipedia <ref>{{Cite pmid|27900053 }}</ref> Receptor tyrosine kinases in carcinogenesis #MMPMID27900053 Zhang XY ; Zhang PY Oncol Lett 2016[Nov]; 12 (5 ): 3679-3682 PMID27900053 show ga Receptor tyrosine kinases (RTKs) are cell surface glycoproteins with enzymatic activity involved in the regulation of various important functions. In all-important physiological functions including differentiation, cell-cell interactions, survival, proliferation, metabolism, migration and signaling these receptors are the key players of regulation. Additionally, mutations of RTKs or their overexpression have been described in many human cancers and are being explored as a novel avenue for a new therapeutic approach. Some of the deregulated RTKs observed to be significantly affected in cancers included vascular endothelial growth factor receptor, epidermal growth factor receptor, fibroblast growth factor receptor, RTK-like orphan receptor 1 (ROR1) and the platelet-derived growth factor receptor. These deregulated RTKs offer attractive possibilities for the new anticancer therapeutic approach involving specific targeting by monoclonal antibodies as well as kinase. The present review aimed to highlight recent perspectives of RTK ROR1 in cancer. äReceptor tyrosine kinases in carcinogenesis (epmc).pdf DeepDyve Pubget Overpricing