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10.1161/CIRCRESAHA.115.301146 http://scihub22266oqcxt.onion/10.1161/CIRCRESAHA.115.301146 C4096686!4096686 !24951762     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24951762 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Circ+Res 2014 ; 115 (1 ): 115-30 Nephropedia Template TP {{tp|p=24951762 |t=2014. Pulmonary arterial hypertension: the clinical syndrome |pdf=|usr=}}{{24951762 }} gab.com Text Pulmonary arterial hypertension: the clinical syndrome JO: Circ Res http://www.kidney.de/pget.php?&tw=1&pmid=24951762 #FOAvip Pulmonary arterial hypertension is a progressive disorder in which endothelial dysfunction and vascular remodeling obstruct small pulmonary arteries, resulting in increased pulmonary vascular resistance and pulmonary pressures. This leads to reduced cardiac output, right heart failure, and ultimately death. In this review, we attempt to answer some important questions commonly asked by patients diagnosed with pulmonary arterial hypertension pertaining to the disease, and aim to provide an explanation in terms of classification, diagnosis, pathophysiology, genetic causes, demographics, and prognostic factors. Furthermore, important molecular pathways that are central to the pathogenesis of pulmonary arterial hypertension are reviewed, including nitric oxide, prostacyclin, endothelin-1, reactive oxygen species, and endothelial and smooth muscle proliferation. Twit Text FOAVip Pulmonary arterial hypertension: the clinical syndrome ????Circ Res http://www.kidney.de/pget.php?&tw=1&pmid=24951762 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24951762 #FOAvip English Wikipedia <ref>{{Cite pmid|24951762 }}</ref> Pulmonary arterial hypertension: the clinical syndrome #MMPMID24951762 Lai YC ; Potoka KC ; Champion HC ; Mora AL ; Gladwin MT Circ Res 2014[Jun]; 115 (1 ): 115-30 PMID24951762 show ga Pulmonary arterial hypertension is a progressive disorder in which endothelial dysfunction and vascular remodeling obstruct small pulmonary arteries, resulting in increased pulmonary vascular resistance and pulmonary pressures. This leads to reduced cardiac output, right heart failure, and ultimately death. In this review, we attempt to answer some important questions commonly asked by patients diagnosed with pulmonary arterial hypertension pertaining to the disease, and aim to provide an explanation in terms of classification, diagnosis, pathophysiology, genetic causes, demographics, and prognostic factors. Furthermore, important molecular pathways that are central to the pathogenesis of pulmonary arterial hypertension are reviewed, including nitric oxide, prostacyclin, endothelin-1, reactive oxygen species, and endothelial and smooth muscle proliferation. |Bone Morphogenetic Protein Receptors, Type II/genetics/physiology [MESH] |Familial Primary Pulmonary Hypertension [MESH] |Female [MESH] |Hemodynamics [MESH] |Humans [MESH] |Hypertension, Pulmonary/*etiology/genetics/mortality/physiopathology [MESH] |Male [MESH] |Mutation [MESH] |Positron-Emission Tomography [MESH] |Prognosis [MESH] |Sex Factors [MESH]Pulmonary arterial hypertension: the clinical syndrome (epmc).pdf DeepDyve Pubget Overpricing