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10.1007/s11906-017-0717-2 http://scihub22266oqcxt.onion/10.1007/s11906-017-0717-2 C5340829!5340829 !28271419     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28271419 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28271419 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Hypertens+Rep 2017 ; 19 (3 ): 20 Nephropedia Template TP {{tp|p=28271419 |t=2017. Pre-eclampsia: the Potential of GSNO Reductase Inhibitors |pdf=|usr=}}{{28271419 }} gab.com Text Pre-eclampsia: the Potential of GSNO Reductase Inhibitors JO: Curr Hypertens Rep http://www.kidney.de/pget.php?&tw=1&pmid=28271419 #FOAvip PURPOSE OF REVIEW: Pre-eclampsia remains a leading worldwide cause of maternal death and of perinatal morbidity. There remains no definitive treatment except delivery of the fetus. RECENT FINDINGS: Recent insights into the cardiovascular changes that are evident prior to, during, and persist after pre-eclampsia have improved understanding of the underlying pathophysiology-disruption of normal endothelial function and decreased nitric oxide bioavailability. S-nitrosoglutathione (GSNO) is an endogenous S-nitrosothiol that acts as a NO pool and, by replenishing or preventing the breakdown of GSNO, endothelial dysfunction can be ameliorated. GSNO reductase inhibitors are a novel class of drug that can increase NO bioavailability. GSNO reductase inhibitors have demonstrated improvement of endothelial dysfunction in animal models, and in vivo human studies have shown them to be well tolerated. GSNOR inhibitors offer a potentially promising option for the management of pre-eclampsia. Twit Text FOAVip Pre-eclampsia: the Potential of GSNO Reductase Inhibitors ????Curr Hypertens Rep http://www.kidney.de/pget.php?&tw=1&pmid=28271419 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28271419 #FOAvip English Wikipedia <ref>{{Cite pmid|28271419 }}</ref> Pre-eclampsia: the Potential of GSNO Reductase Inhibitors #MMPMID28271419 Everett TR ; Wilkinson IB ; Lees CC Curr Hypertens Rep 2017[Mar]; 19 (3 ): 20 PMID28271419 show ga PURPOSE OF REVIEW: Pre-eclampsia remains a leading worldwide cause of maternal death and of perinatal morbidity. There remains no definitive treatment except delivery of the fetus. RECENT FINDINGS: Recent insights into the cardiovascular changes that are evident prior to, during, and persist after pre-eclampsia have improved understanding of the underlying pathophysiology-disruption of normal endothelial function and decreased nitric oxide bioavailability. S-nitrosoglutathione (GSNO) is an endogenous S-nitrosothiol that acts as a NO pool and, by replenishing or preventing the breakdown of GSNO, endothelial dysfunction can be ameliorated. GSNO reductase inhibitors are a novel class of drug that can increase NO bioavailability. GSNO reductase inhibitors have demonstrated improvement of endothelial dysfunction in animal models, and in vivo human studies have shown them to be well tolerated. GSNOR inhibitors offer a potentially promising option for the management of pre-eclampsia. |Animals [MESH] |Endothelium/drug effects/metabolism [MESH] |Enzyme Inhibitors/*therapeutic use [MESH] |Female [MESH] |Humans [MESH] |Hypertension/drug therapy [MESH] |Nitric Oxide/metabolism [MESH] |Pre-Eclampsia/*drug therapy [MESH] |Pregnancy [MESH]Pre-eclampsia: the Potential of GSNO Reductase Inhibitors (epmc).pdf DeepDyve Pubget Overpricing