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10.1186/s40591-014-0037-8 http://scihub22266oqcxt.onion/10.1186/s40591-014-0037-8 C4451966!4451966 !26056601     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26056601 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26056601 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mol+Cell+Ther 2014 ; 2 (ä): 37 Nephropedia Template TP {{tp|p=26056601 |t=2014. Platelet dysfunction in injured patients |pdf=|usr=}}{{26056601 }} gab.com Text Platelet dysfunction in injured patients JO: Mol Cell Ther http://www.kidney.de/pget.php?&tw=1&pmid=26056601 #FOAvip A renewed understanding of Trauma Induced Coagulopathy (TIC) has implicated platelets as a crucial mediator and potential therapeutic target in hemostasis. While the importance of abnormal coagulation tests is well described in trauma, there is a paucity of data regarding the role of platelets in coagulopathy. New coagulation models, namely the cell-based-model of hemostasis, have refocused attention toward the platelet and endothelium as key regulators of clot formation. Although platelet dysfunction has been associated with worse outcomes in trauma, the mechanisms which platelet dysfunction contributes to coagulopathy are poorly understood. The goal of this review article is to outline recent advances in understanding hemostasis and the ensuing cellular dysfunction that contributes to the exsanguination of a critically injured patient. Twit Text FOAVip Platelet dysfunction in injured patients ????Mol Cell Ther http://www.kidney.de/pget.php?&tw=1&pmid=26056601 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26056601 #FOAvip English Wikipedia <ref>{{Cite pmid|26056601 }}</ref> Platelet dysfunction in injured patients #MMPMID26056601 Saillant NN ; Sims CA Mol Cell Ther 2014[]; 2 (ä): 37 PMID26056601 show ga A renewed understanding of Trauma Induced Coagulopathy (TIC) has implicated platelets as a crucial mediator and potential therapeutic target in hemostasis. While the importance of abnormal coagulation tests is well described in trauma, there is a paucity of data regarding the role of platelets in coagulopathy. New coagulation models, namely the cell-based-model of hemostasis, have refocused attention toward the platelet and endothelium as key regulators of clot formation. Although platelet dysfunction has been associated with worse outcomes in trauma, the mechanisms which platelet dysfunction contributes to coagulopathy are poorly understood. The goal of this review article is to outline recent advances in understanding hemostasis and the ensuing cellular dysfunction that contributes to the exsanguination of a critically injured patient. äPlatelet dysfunction in injured patients (epmc).pdf DeepDyve Pubget Overpricing