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10.1091/mbc.E16-01-0002 http://scihub22266oqcxt.onion/10.1091/mbc.E16-01-0002 C4814223!4814223!26864629     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26864629&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26864629.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mol+Biol+Cell 2016 ; 27 (7): 1170-80 Nephropedia Template TP {{tp|p=26864629|t=2016. Phosphoinositide kinase signaling controls ER-PM cross-talk |pdf=|usr=}}{{26864629}} gab.com Text Phosphoinositide kinase signaling controls ER-PM cross-talk JO: Mol Biol Cell http://www.kidney.de/pget.php?&tw=1&pmid=26864629 #FOAvip Phosphoinositide kinase signaling controls a conserved PDK-TORC2-Akt signaling cascade as part of a homeostasis network that allows the ER to modulate essential responses, including Ca2+-regulated lipid biogenesis, upon plasma membrane (PM) stress. Loss of ER-PM junctions impairs this protective response, leading to PM integrity defects upon heat stress. Twit Text FOAVip Phosphoinositide kinase signaling controls ER-PM cross-talk ????Mol Biol Cell http://www.kidney.de/pget.php?&tw=1&pmid=26864629 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26864629 #FOAvip English Wikipedia <ref>{{Cite pmid|26864629}}</ref> Phosphoinositide kinase signaling controls ER-PM cross-talk #MMPMID26864629 Omnus DJ; Manford AG; Bader JM; Emr SD; Stefan CJ Mol Biol Cell 2016[Apr]; 27 (7): 1170-80 PMID26864629show ga Phosphoinositide kinase signaling controls a conserved PDK-TORC2-Akt signaling cascade as part of a homeostasis network that allows the ER to modulate essential responses, including Ca2+-regulated lipid biogenesis, upon plasma membrane (PM) stress. Loss of ER-PM junctions impairs this protective response, leading to PM integrity defects upon heat stress. äPhosphoinositide kinase signaling controls ER-PM cross-talk (epmc).pdf DeepDyve Pubget Overpricing