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10.1016/j.ccep.2016.02.003 http://scihub22266oqcxt.onion/10.1016/j.ccep.2016.02.003 C4893809!4893809 !27261829     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27261829 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Card+Electrophysiol+Clin 2016 ; 8 (2 ): 385-93 Nephropedia Template TP {{tp|p=27261829 |t=2016. Pharmacogenetics of Potassium Channel Blockers |pdf=|usr=}}{{27261829 }} gab.com Text Pharmacogenetics of Potassium Channel Blockers JO: Card Electrophysiol Clin http://www.kidney.de/pget.php?&tw=1&pmid=27261829 #FOAvip The QT interval on surface electrocardiograms provides a model of a multicomponent integrated readout of many biological systems, including ion channels, modulatory subunits, signaling systems that modulate their activity, and mechanisms that regulate the expression of their responsible genes. The problem of drug exposure causing exaggerated QT interval prolongation and torsades de pointes highlights the multicomponent nature of cardiac repolarization and the way in which simple perturbations can yield exaggerated responses. Future directions will involve cellular approaches coupled to evolving technologies that can interrogate multicellular systems and provide a sophisticated view of mechanisms in this previously idiosyncratic drug reaction. Twit Text FOAVip Pharmacogenetics of Potassium Channel Blockers ????Card Electrophysiol Clin http://www.kidney.de/pget.php?&tw=1&pmid=27261829 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27261829 #FOAvip English Wikipedia <ref>{{Cite pmid|27261829 }}</ref> Pharmacogenetics of Potassium Channel Blockers #MMPMID27261829 Roden DM Card Electrophysiol Clin 2016[Jun]; 8 (2 ): 385-93 PMID27261829 show ga The QT interval on surface electrocardiograms provides a model of a multicomponent integrated readout of many biological systems, including ion channels, modulatory subunits, signaling systems that modulate their activity, and mechanisms that regulate the expression of their responsible genes. The problem of drug exposure causing exaggerated QT interval prolongation and torsades de pointes highlights the multicomponent nature of cardiac repolarization and the way in which simple perturbations can yield exaggerated responses. Future directions will involve cellular approaches coupled to evolving technologies that can interrogate multicellular systems and provide a sophisticated view of mechanisms in this previously idiosyncratic drug reaction. |*Arrhythmias, Cardiac/drug therapy/genetics [MESH] |Animals [MESH] |Humans [MESH] |Mice [MESH] |Pharmacogenetics [MESH]Pharmacogenetics of Potassium Channel Blockers (epmc).pdf DeepDyve Pubget Overpricing