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10.1007/s12178-015-9277-8 http://scihub22266oqcxt.onion/10.1007/s12178-015-9277-8 C4596210!4596210 !26142896     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26142896 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26142896 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Rev+Musculoskelet+Med 2015 ; 8 (3 ): 201-9 Nephropedia Template TP {{tp|p=26142896 |t=2015. Pathophysiology and risk factors for osteonecrosis |pdf=|usr=}}{{26142896 }} gab.com Text Pathophysiology and risk factors for osteonecrosis JO: Curr Rev Musculoskelet Med http://www.kidney.de/pget.php?&tw=1&pmid=26142896 #FOAvip Osteonecrosis, also known as avascular necrosis or AVN, is characterized by a stereotypical pattern of cell death and a complex repair process of bone resorption and formation. It is not the necrosis itself but rather the resorptive component of the repair process that results in loss of structural integrity and subchondral fracture. Most likely, a common pathophysiological pathway exists involving compromised subchondral microcirculation. Decreased femoral head blood flow can occur through three mechanisms: vascular interruption by fractures or dislocation, intravascular occlusion from thrombi or embolic fat, or intraosseous extravascular compression from lipocyte hypertrophy or Gaucher cells. In this review, we emphasize etiologic relationships derived mostly from longitudinal cohort studies or meta-analyses whose causal relationships to osteonecrosis can be estimated with confidence. Understanding risk factors and pathophysiology has therapeutic implications since several treatment regimens are available to optimize femoral head circulation, interrupt bone resorption, and preserve the subchondral bone. Twit Text FOAVip Pathophysiology and risk factors for osteonecrosis ????Curr Rev Musculoskelet Med http://www.kidney.de/pget.php?&tw=1&pmid=26142896 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26142896 #FOAvip English Wikipedia <ref>{{Cite pmid|26142896 }}</ref> Pathophysiology and risk factors for osteonecrosis #MMPMID26142896 Shah KN ; Racine J ; Jones LC ; Aaron RK Curr Rev Musculoskelet Med 2015[Sep]; 8 (3 ): 201-9 PMID26142896 show ga Osteonecrosis, also known as avascular necrosis or AVN, is characterized by a stereotypical pattern of cell death and a complex repair process of bone resorption and formation. It is not the necrosis itself but rather the resorptive component of the repair process that results in loss of structural integrity and subchondral fracture. Most likely, a common pathophysiological pathway exists involving compromised subchondral microcirculation. Decreased femoral head blood flow can occur through three mechanisms: vascular interruption by fractures or dislocation, intravascular occlusion from thrombi or embolic fat, or intraosseous extravascular compression from lipocyte hypertrophy or Gaucher cells. In this review, we emphasize etiologic relationships derived mostly from longitudinal cohort studies or meta-analyses whose causal relationships to osteonecrosis can be estimated with confidence. Understanding risk factors and pathophysiology has therapeutic implications since several treatment regimens are available to optimize femoral head circulation, interrupt bone resorption, and preserve the subchondral bone. äPathophysiology and risk factors for osteonecrosis (epmc).pdf DeepDyve Pubget Overpricing