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10.2176/nmc.ra.2016-0051 http://scihub22266oqcxt.onion/10.2176/nmc.ra.2016-0051 C4908075!4908075 !27076383     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27076383 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Neurol+Med+Chir+(Tokyo) 2016 ; 56 (6 ): 317-25 Nephropedia Template TP {{tp|p=27076383 |t=2016. Pathogenesis of Brain Arteriovenous Malformations |pdf=|usr=}}{{27076383 }} gab.com Text Pathogenesis of Brain Arteriovenous Malformations JO: Neurol Med Chir (Tokyo) http://www.kidney.de/pget.php?&tw=1&pmid=27076383 #FOAvip Brain arteriovenous malformations (bAVMs) represent a high risk of intracranial hemorrhages, which are substantial causes of morbidity and mortality of bAVMs, especially in children and young adults. Although a variety of factors leading to hemorrhages of bAVMs are investigated extensively, their pathogenesis is still not well elucidated. The author has reviewed the updated data of genetic aspects of bAVMs, especially focusing on clinical and experimental knowledge from hereditary hemorrhagic telangiectasia, which is the representative genetic disease presenting with bAVMs caused by loss-of-function in one of the two genes: endoglin and activin receptor-like kinase 1. This knowledge may allow us to infer the pathogensis of sporadic bAVMs and in the development of new medical therapies for them. Twit Text FOAVip Pathogenesis of Brain Arteriovenous Malformations ????Neurol Med Chir (Tokyo) http://www.kidney.de/pget.php?&tw=1&pmid=27076383 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27076383 #FOAvip English Wikipedia <ref>{{Cite pmid|27076383 }}</ref> Pathogenesis of Brain Arteriovenous Malformations #MMPMID27076383 Komiyama M Neurol Med Chir (Tokyo) 2016[Jun]; 56 (6 ): 317-25 PMID27076383 show ga Brain arteriovenous malformations (bAVMs) represent a high risk of intracranial hemorrhages, which are substantial causes of morbidity and mortality of bAVMs, especially in children and young adults. Although a variety of factors leading to hemorrhages of bAVMs are investigated extensively, their pathogenesis is still not well elucidated. The author has reviewed the updated data of genetic aspects of bAVMs, especially focusing on clinical and experimental knowledge from hereditary hemorrhagic telangiectasia, which is the representative genetic disease presenting with bAVMs caused by loss-of-function in one of the two genes: endoglin and activin receptor-like kinase 1. This knowledge may allow us to infer the pathogensis of sporadic bAVMs and in the development of new medical therapies for them. |Humans [MESH]Pathogenesis of Brain Arteriovenous Malformations (epmc).pdf DeepDyve Pubget Overpricing