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10.3389/fphys.2017.00600 http://scihub22266oqcxt.onion/10.3389/fphys.2017.00600 C5572357!5572357 !28878685     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28878685 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Front+Physiol 2017 ; 8 (ä): 600 Nephropedia Template TP {{tp|p=28878685 |t=2017. Oxidative Stress-Mediated Atherosclerosis: Mechanisms and Therapies |pdf=|usr=}}{{28878685 }} gab.com Text Oxidative Stress-Mediated Atherosclerosis: Mechanisms and Therapies JO: Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=28878685 #FOAvip Atherogenesis, the formation of atherosclerotic plaques, is a complex process that involves several mechanisms, including endothelial dysfunction, neovascularization, vascular proliferation, apoptosis, matrix degradation, inflammation, and thrombosis. The pathogenesis and progression of atherosclerosis are explained differently by different scholars. One of the most common theories is the destruction of well-balanced homeostatic mechanisms, which incurs the oxidative stress. And oxidative stress is widely regarded as the redox status realized when an imbalance exists between antioxidant capability and activity species including reactive oxygen (ROS), nitrogen (RNS) and halogen species, non-radical as well as free radical species. This occurrence results in cell injury due to direct oxidation of cellular protein, lipid, and DNA or via cell death signaling pathways responsible for accelerating atherogenesis. This paper discusses inflammation, mitochondria, autophagy, apoptosis, and epigenetics as they induce oxidative stress in atherosclerosis, as well as various treatments for antioxidative stress that may prevent atherosclerosis. Twit Text FOAVip Oxidative Stress-Mediated Atherosclerosis: Mechanisms and Therapies ????Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=28878685 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28878685 #FOAvip English Wikipedia <ref>{{Cite pmid|28878685 }}</ref> Oxidative Stress-Mediated Atherosclerosis: Mechanisms and Therapies #MMPMID28878685 Yang X ; Li Y ; Li Y ; Ren X ; Zhang X ; Hu D ; Gao Y ; Xing Y ; Shang H Front Physiol 2017[]; 8 (ä): 600 PMID28878685 show ga Atherogenesis, the formation of atherosclerotic plaques, is a complex process that involves several mechanisms, including endothelial dysfunction, neovascularization, vascular proliferation, apoptosis, matrix degradation, inflammation, and thrombosis. The pathogenesis and progression of atherosclerosis are explained differently by different scholars. One of the most common theories is the destruction of well-balanced homeostatic mechanisms, which incurs the oxidative stress. And oxidative stress is widely regarded as the redox status realized when an imbalance exists between antioxidant capability and activity species including reactive oxygen (ROS), nitrogen (RNS) and halogen species, non-radical as well as free radical species. This occurrence results in cell injury due to direct oxidation of cellular protein, lipid, and DNA or via cell death signaling pathways responsible for accelerating atherogenesis. This paper discusses inflammation, mitochondria, autophagy, apoptosis, and epigenetics as they induce oxidative stress in atherosclerosis, as well as various treatments for antioxidative stress that may prevent atherosclerosis. äOxidative Stress-Mediated Atherosclerosis: Mechanisms and Therapies (epmc).pdf DeepDyve Pubget Overpricing