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10.1016/j.coviro.2012.07.001 http://scihub22266oqcxt.onion/10.1016/j.coviro.2012.07.001 C4617531!4617531 !22858118     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=22858118 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\22858118 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Opin+Virol 2012 ; 2 (4 ): 453-8 Nephropedia Template TP {{tp|p=22858118 |t=2012. Novel mechanisms of EBV-induced oncogenesis |pdf=|usr=}}{{22858118 }} gab.com Text Novel mechanisms of EBV-induced oncogenesis JO: Curr Opin Virol http://www.kidney.de/pget.php?&tw=1&pmid=22858118 #FOAvip Epstein-Barr virus is an etiologic factor in multiple types of cancer that primarily develop in lymphocytes and epithelial cells. The tumors are latently infected yet express distinct subsets of viral proteins that are essential for transformation. The viral oncogenes may be expressed in a subset of cells and are transferred through exosomes to many cells to induce growth and alter the tumor environment. In some of the viral cancers, viral proteins are not expressed, however, the viral miRNAs can alter growth by decreasing expression of negative regulators of cell growth such as tumor suppressors and cellular proteins that induce apoptosis. Twit Text FOAVip Novel mechanisms of EBV-induced oncogenesis ????Curr Opin Virol http://www.kidney.de/pget.php?&tw=1&pmid=22858118 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22858118 #FOAvip English Wikipedia <ref>{{Cite pmid|22858118 }}</ref> Novel mechanisms of EBV-induced oncogenesis #MMPMID22858118 Raab-Traub N Curr Opin Virol 2012[Aug]; 2 (4 ): 453-8 PMID22858118 show ga Epstein-Barr virus is an etiologic factor in multiple types of cancer that primarily develop in lymphocytes and epithelial cells. The tumors are latently infected yet express distinct subsets of viral proteins that are essential for transformation. The viral oncogenes may be expressed in a subset of cells and are transferred through exosomes to many cells to induce growth and alter the tumor environment. In some of the viral cancers, viral proteins are not expressed, however, the viral miRNAs can alter growth by decreasing expression of negative regulators of cell growth such as tumor suppressors and cellular proteins that induce apoptosis. |*Cell Transformation, Viral [MESH] |Animals [MESH] |Cell Proliferation [MESH] |Gene Expression Regulation, Viral [MESH] |Herpesvirus 4, Human/genetics/*physiology [MESH] |Humans [MESH] |Neoplasms/genetics/metabolism/physiopathology/*virology [MESH]Novel mechanisms of EBV-induced oncogenesis (epmc).pdf DeepDyve Pubget Overpricing