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10.1002/path.4775 http://scihub22266oqcxt.onion/10.1002/path.4775 C5071153!5071153!27477320     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27477320&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27477320.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Pathol 2016 ; 240 (3): 282-90 Nephropedia Template TP {{tp|p=27477320|t=2016. NTRK3 kinase fusions in Spitz tumours |pdf=|usr=}}{{27477320}} gab.com Text NTRK3 kinase fusions in Spitz tumours JO: J Pathol http://www.kidney.de/pget.php?&tw=1&pmid=27477320 #FOAvip Oncogenic fusions in TRK family receptor tyrosine kinases have been identified in several cancers and can serve as therapeutic targets. We identified ETV6-NTRK3, MYO5A-NTRK3 and MYH9-NTRK3 fusions in Spitz tumours and demonstrate that NTRK3 fusions constitutively activate the MAPK, PI3K and PLC?1 pathways in melanocytes. This signalling was inhibited by DS-6051a, a small molecule inhibitor of NTRK1/2/3 and ROS1. NTRK3 fusions expand the range of oncogenic kinase fusions in melanocytic neoplasms and offer targets for a small subset of melanomas for which currently no targeted options exist. Twit Text FOAVip NTRK3 kinase fusions in Spitz tumours ????J Pathol http://www.kidney.de/pget.php?&tw=1&pmid=27477320 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27477320 #FOAvip English Wikipedia <ref>{{Cite pmid|27477320}}</ref> NTRK3 kinase fusions in Spitz tumours #MMPMID27477320 Yeh I; Tee MK; Botton T; Shain AH; Sparatta AJ; Gagnon A; Vemula SS; Garrido MC; Nakamaru K; Isoyama T; McCalmont TH; LeBoit PE; Bastian BC J Pathol 2016[Nov]; 240 (3): 282-90 PMID27477320show ga Oncogenic fusions in TRK family receptor tyrosine kinases have been identified in several cancers and can serve as therapeutic targets. We identified ETV6-NTRK3, MYO5A-NTRK3 and MYH9-NTRK3 fusions in Spitz tumours and demonstrate that NTRK3 fusions constitutively activate the MAPK, PI3K and PLC?1 pathways in melanocytes. This signalling was inhibited by DS-6051a, a small molecule inhibitor of NTRK1/2/3 and ROS1. NTRK3 fusions expand the range of oncogenic kinase fusions in melanocytic neoplasms and offer targets for a small subset of melanomas for which currently no targeted options exist. äNTRK3 kinase fusions in Spitz tumours (epmc).pdf DeepDyve Pubget Overpricing